Yuling Chow

Will HK be considering adding Novavax as a choice of COVID-19 vaccine, seeing how it’s on par with mRNA in efficacy, has potentially greater durability, and may also allay vaccine hesitancy and boost uptake by virtue of it being a non-mRNA vaccine? @HKU_SPH @bencowling88

Why do #COVID & #flu hit older adults harder? 👉 It’s not just weaker immunity — aging lung tissue actively drives excessive inflammation. ➡️ Key mechanism: Aging lung fibroblasts trigger NF-κB–mediated “inflammaging”, recruiting dysfunctional immune cells. 👉 The problem is overreaction, not underreaction. ➡️ Researchers identified GZMK+ T cells in inflamed lung clusters— seen in severe COVID 👉 These cells fail to control infection but still damage lungs. ➡️ Striking finding: Young lungs engineered with aging signals behaved like “old lungs” under infection 👉 Severe disease is driven by tissue biology, not just age. 1/

From a former newspaper journalist:

@SketchesbyBoze The future belongs to people who read widely, think critically, AND protect themselves from the SARS-CoV-2 virus that causes cognitive damage. I mask during the ongoing pandemic because I read the scientific literature. Reading and masking go hand in hand.

Why might insulin resistance go up after COVID? Probably not because of one single cause, but because of a whole network of inflammatory and metabolic changes. A new narrative review.🧵

It's been a very long known fact that weakened immune systems are a perfect breeding ground for drug resistance. Graphs like this are *zero surprise* to me. Drug resistant gonorrhoaea on the charge:

The WHO told the world 6 years ago today that "COVID-19 is NOT airborne." This is disinformation. They have not deleted the post and have not apologized for the tens of millions of lives lost and hundreds of millions of people now with #LongCOVID because of their misguidance.

Invasive meningococcal disease rebound in older adults post-COVID-19 pandemic sciencedirect.com/science/articl…

"Even Mild COVID Cases Leave Lasting Brain Changes in Young Adults"

@NSWHealth you great numpties. Measles is AIRBORNE. Also one of the most infectious diseases that exists. An infected person just *breathing* is enough to spread it. They don't need to be "coughing or sneezing" And it can stay in the air (e.g. in a waiting room) for up to 2 hours

Here’s a little more detail from Prof Beggs’ testimony where he again confirms that the BULK of the SARS-CoV-2 virus is carried in the small airborne aerosols, NOT in the larger droplets which rapidly fall to the ground. This is CRUCIAL for infection control purposes.

The Human Cost of Failing to Name COVID ‘Airborne’ "It’s difficult to know why the word “airborne” is avoided so consistently, but the impact is clear: without that word, occupational health and safety requirements can be evaded." #COVIDisAirBorne thetyee.ca/Analysis/2026/…

Invasive meningococcal disease rebound in older adults post-COVID-19 p... sciencedirect.com/science/articl…

Explain to me how I’m supposed to respond to a parent who says that 4️⃣ college grads acquired POTS in Dec 2025 & 2️⃣ are bed-bound. That’s not “continue to live with”… that’s “just got because of accumulating damage from failing to mitigate a highly dangerous airborne pathogen.”

Public health bodies didn't warn against smoking till *decades after* scientists showed smoking harmed people. Listen to the scientists, not public health. "If you’re doing what you can to avoid (re)infection, your behaviour is 100% aligned with best current available evidence."

Science takes ~17 years to reach clinical practice. The science in 2026 is clear: we don’t have another decade to wait on SARS-CoV-2. If you’re doing what you can to avoid (re)infection, your behaviour is 100% aligned with best current available evidence.

1) 🇩🇪 This German study found that Long Covid patients (n= 128) had less circulating, cell-free mitochondrial DNA than recovered controls (n = 100).

How much of the scientific literature is just straight up lies? 30% 50% 70%? 50-70% would be my guess. In this meta-analysis of CBT for MECFS: *PEM, the main characteristic of the disease, was not required for diagnosis in any of the included studies (because duh....they know ahead of time if it was they would not get the results they want). *Patients remained severely ill and severely disabled after CBT. (Right...so what worked? What improved?) *Quality of life, which was one of the two objectives according to the registration of the meta-analysis, did not improve. (OK...so how did they spin this????) *Different forms of CBT did not lead to objective improvement, and an extensive review of the literature found that CBT has a negative instead of a positive effect on work and illness benefit status. (But again....somehow they spun a different story???? Crazy making). Yet Kolala et al. conclusion was that although CBT did not lead to statistically significant improvement in fatigue - individual face-to-face CBT had a large effect on reducing fatigue - self-directed CBT had a large effect on increasing physical functioning. Its insane. Its just lying. You can fancy up what it is called - data manipulation, statistical massage, sub-group analysis.....its lying. They know they are lying. Thankful for Dr. Mark Vink and other scientists who keep fighting for the truth and keep spending their valuable time on tearing down bad science and lies. What we read is mostly lies - I really believe this.

SARS-CoV-2 Spike protein can reach the brain through multiple routes — more than most viruses. A clear summary of known pathways: 1. Neuronal/axonal routes: • Olfactory nerve (nose → olfactory bulb → brain) • Trigeminal nerve (face/nose → brainstem) • Vagus nerve (lung/gut → brainstem) 2. Hematogenous route (via blood): • Crossing or disrupting the blood-brain barrier (BBB) Key facilitators: - Receptors: ACE2 (primary), NRP-1 (strong booster, especially in olfactory epithelium & astrocytes), CD147, AXL - Attachment factors: Gangliosides (e.g., GM1, GM3 — sialic acid binding in lipid rafts, highly abundant in brain) - BBB disruption: Spike S1 activates RhoA → tight junction disassembly (ZO-1, Claudin-5) → increased permeability, even without full virus replication. This broad combination helps explain neurological symptoms and potential long-term effects. Early studies focused on ACE2 + olfactory route. Later work added NRP-1 (binds furin-cleaved Spike), ganglioside GM1 in cerebrovascular cells, and direct Spike effects on tight junctions via RhoA signaling — even without full virus replication. Similar mechanisms seen in other neurotropic viruses (HSV, flaviviruses), but the combo here (respiratory entry + multiple cranial nerves + BBB leak + alternative receptors) is particularly notable. Circulating Spike alone can contribute to barrier issues. #NeuroCovid Sources & further reading below.

Australian National University researchers argue Parkinsons may start in the gut, where microbiome changes may damage a protective cell coating and disrupt ion balance, linking the gut, blood vessels and brain as one connected disease. sciencedirect.com/science/articl…

Thanks to @TIME & @vero_greenwood for highlighting a massive unmet medical need: People who get sick with a virus and don't get better. A growing body of research is revealing a pattern: →an immune trigger →a shift in immune and neural signaling →a self-reinforcing neuroimmune loop across systems Post-viral illness is one expression. →other immune triggers are likely part of the same biology. #Neuroimmune science is critical to more deeply understanding and treating complex chronic disorders like #LONGCOVID, #MECFS & #POTS. It's multisystemic and looks at both the trigger and the downstream cascade of debilitating symptoms. #PatientsCantWait time.com/article/2026/0…