Sagar ☀️

Highest IPL totals at these venues: 🏟️ Chinnaswamy — 287/3: SRH 🏟️ Uppal — 286/6: SRH 🏟️ Arun Jaitley — 278/3: SRH 🏟️ Sawai Mansingh — 229: GT & SRH ( joint highest ) 🏟️ Wankhede — 249/4: SRH We don’t play on flat pitches. We make pitches look flat. 🔥

Many many happy returns of the day @s4__sanjay 🎉🎁🎈🎂🎊

People make fun of cave divers, and fair enough it is an absurdly dangerous pursuit but without people willing to pursue something that extreme you would never have the specialists capable of performing cave rescues. The two divers who found the thirteen boys trapped inside a flooded cave in Thailand had to navigate four kilometers of passageways, underwater. If I was one of those kids I’d be forever grateful some madman would do this.

Nah, esto es pura inteligencia futbolística 🧠⚽

🙌

If you don’t actively shape the life you desire each day, you’ll eventually wake up to find that years have slipped by unnoticed.

Koçu, nasıl bir canavar yarattığını fark ediyor:

Em shot ra maama! 🤌🤌🤌

¡Esta cosa debería estar prohibida!

Man find canister labeled 'Nazi red mercury' which appears to burn metal without heat. 🧐

Shocking beyond words .. Share max . Let this reach all.. A ₹1.6 crore hospital bill collapsed to just ₹27 lakhs, not because of a lawyer, not because of connections, but because of an AI chatbot. This family uploaded the entire medical bill into an AI system and let it audit every line item. The AI detected duplicate billing, illegal code stacking, and procedures that hospitals are not legally allowed to charge for. It then drafted a legally structured dispute letter referencing exact compliance violations. The hospital had no counter. The bill was slashed by over eighty percent. This is the real power of AI in healthcare — medical bill audits, hospital compliance checks, insurance dispute automation, and patient protection at scale.

@RandomCricketP1 The Answer is None. Klaasen is the 1st & only batter in IPL history to score 600+ runs without batting at top 3 spots in the entire season. #SRH #IPL2026

Klaasen has such a cold look every time the camera zooms in on his face after he has hit a marvellous six. Proper in the zone look. Also since he has crossed 600 today, can anyone tell how many non top 3 batters have scored 600+ in the league's history? Curious.

@SunRisers Please don't impact head, make him field every match and give him the ball atleast 1 over to kill the momentum..... 🥺🥺🙏🙏🙏

Came, bowled and walked away with a wicket 😎

What a beautiful explanation of Entropy. Too good is this girl’s clarity and understanding of spirituality & philosophy

Computer programming and peacock feathers.

Take too much Ozempic, and your brain stops wanting things: food, sex, even the urge to get out of bed. People end up in hospital beds for days, staring at the ceiling, feeling nothing. The medical name for that state is anhedonia, and it tells you how the drug actually works. Ozempic, Wegovy, and Mounjaro all belong to the same drug family, called GLP-1s. They kill hunger. They also quiet almost every other craving your brain produces. Inside your brain there is a small region that makes a chemical called dopamine. Dopamine is your brain’s “this is worth wanting” chemical, the reason you reach for one more bite of pasta, refresh your inbox one more time, or pick up your phone every few minutes. GLP-1 drugs reach that region and turn the dopamine down. The right dose dampens the loudest craving first: food. Take too much, and the volume drops on everything else, sex, exercise, work, even the urge to get out of bed in the morning. Anhedonia is the medical name for not feeling pleasure from anything at all. It looks identical to deep depression. The good news is that anhedonia from GLP-1s has an off switch: once the drug clears your system, the wanting comes back. The FDA has logged over 1,150 reports of bad reactions tied to compounded GLP-1s through July 2025. These are custom-mixed versions made by smaller pharmacies. In many of those cases, patients accidentally took five to twenty times their prescribed dose. The cause is usually confusion between milliliters and units when measuring out a dose with an insulin syringe, since compounded versions come in plain vials instead of the pre-filled pens that brand-name Ozempic uses. About 15 million Americans currently use a GLP-1, roughly one in eight adults. Around 75% of them eventually quit. Cost and side effects are the top reasons. A growing number describe a third reason that patients call “the lights dimming,” a flat, gray feeling across the whole day that doctors now recognize as anhedonia caused by the drug itself. This same mechanism has caught pharma’s attention. Eli Lilly is now running two large clinical trials with a combined 2,200 patients to see if a GLP-1 drug can treat alcohol addiction. The bet is that the same brain switch that turns off cravings for food can also turn off cravings for alcohol, cocaine, nicotine, and gambling. A 2026 psychiatry review put it bluntly: doctors should be treating these as psychiatric drugs, because that is what they have turned out to be. The drug works by quieting your brain’s signal that something is worth wanting. A normal dose turns the volume down on food cravings. Push the dose too high, and everything else goes quiet too.

@GiorgiaMeloni @HarshaSRHian Waah Modi ji 🌝🌝 #Melodi #Melody

Thank you for the gift

水の中に沈めた鏡で、虹をつくる🌈

His name was Yellapragada Subbarao. He was born on January 12 1895 in Bhimavaram, in present-day Andhra Pradesh. His family was poor. Several of his siblings died young from disease. He studied at Madras Medical College but his British professor deliberately gave him a lesser diploma instead of a full MBBS degree. He scraped together enough money and sailed to America in 1923. He arrived in Boston with almost nothing. To pay his fees at Harvard Medical School he worked as an attendant at a hospital, cleaning rooms and changing bedsheets at night. Colleagues called him the Indian who cleans toilets. He did not stop. At Harvard he began research with chemist Cyrus Fiske. Together they developed the Fiske-Subbarao method for measuring phosphorus in body fluids, still used in biochemistry today. He then discovered the role of phosphocreatine and adenosine triphosphate in muscular activity. That discovery entered biochemistry textbooks worldwide. It is what we now call ATP, the energy currency of every living cell. Harvard denied him a full professorship. He was a foreigner and had few friends in the right circles. His colleague Cyrus Fiske suppressed and destroyed many of his contributions out of jealousy. Years of Subbarao’s work had to be rediscovered by other scientists because Fiske would not let them be published. He joined Lederle Laboratories instead. There he developed the first method to synthesise folic acid, Vitamin B9. He showed it could treat megaloblastic anaemia and tropical sprue. He then helped develop methotrexate, one of the first chemotherapy drugs, still used today to treat cancer and rheumatoid arthritis. He led the team that discovered Aureomycin, the first tetracycline antibiotic, more powerful than penicillin, which saved hundreds of thousands of lives during and after World War II. He never became an American citizen. He lived in the United States for 25 years on a temporary visa. He applied for permanent residence and never received it. On August 8 1948 he died of cardiac arrest in New York. He was 53 years old. No citizenship. No Nobel Prize. No fame. A writer named Doron Antrim wrote this about him in 1950. “You have probably never heard of Dr. Yellapragada Subbarao. Yet because he lived, you may be alive today.” The drug that treated your anaemia. The antibiotic that fought your infection. The chemotherapy that gave someone more time. All of it traces back to a man from Bhimavaram who cleaned hospital rooms to pay his Harvard fees. India forgot him. Science did not. Follow for stories India deserves to remember.

The greatest set-up of all time.. 🏏