Greta Fox @theWHN #MaskFairy #NoMaskBans

I have a dream. It's not a big dream. It's just a little dream & I hope you don't find this too crazy is that I would like the people of this community to feel that if God forbid there was a medical emergency that going to the hospital would actually be a WISE thing to do.

This is great news! Now we need to show that these accumulations are amyloid in nature.

@Gmwetz @dbkell No this is a cell-based whole blood analysis. Any lab with a flow cytometer can do it.

1/7) We are excited to share our preprint, where we present a method to measure thrombo-inflammation that lead to systemic thrombotic endothelialitis in Long COVID, other post-viral and inflammatory diseases in general: biorxiv.org/content/10.648… with @dbkell and lead author Anél Thompson

What I love about this picture is that the high quality mask is a non issue. You can see he’s smiling. He’s clearly not anxious. He’s out and about in the community. He understands technology helps preserve health. It’s strategic masking & it should be embraced.

Perhaps identifying the wrong mode of transmission (for all healthcare in the UK) and recommending the wrong precautions comes under "Serious Incident" rather than "Never Event" I think it should be both...

What I’d NEVER do as a brain injury rehab doctor: • Motorcycle • Tackle football (for my kids) • Ignore BP • Skip helmets • Normalize repeat COVID infections Because I take care of what happens next.

@BarryHunt008 @WHO AND STOP MEASURING SEVERITY BY THE ACUTE PHASE ONLY!!!

Dear .@WHO COVID IS A SYSTEMIC IMMUNODESTRUCTIVE ONCOGENIC DISEASE STOP CALLING COVID A RESPIRATORY DISEASE STOP LUMPING COVID IN WITH RESPIRATORY DISEASES STOP NORMALIZING COVID STOP MINIMIZING COVID

You got the -Mode of Transmission wrong -AGPs wrong -precautions wrong You failed to involve/listen to experts You had no oversight You provided inadequate PPE You exposed people to infections Your erroneous beliefs caused harm & you kept doing it Inq rpt - don't do that again

This is the reality of modeling aerosols all day🤢 It's just a lot nicer not to think about that, or be like..."nah all the bioaerosols kind of just die on their own in the few seconds between when they are exhaled by someone else and I inhale them..." Because otherwise?🤮

Please do read the mega thread.

I hope they get transmission right. I hope they start some soul searching amongst those who found evidence for droplets that was not there. And perhaps then the NIPCMs which still advocate a mode of transmission that "defies physics" will change the bloody manuals.

Covid Response - Epic Fail

It is not an understatement to say that I hyperventilated a little when I opened this guide. Incredible resource!!!

The pandemic has killed over 30 million–and left more than 400 million suffering from Long COVID. It is one of the most catastrophic events in modern history and it is not over - The COVID-19 pandemic at 6 years: Mass death, debilitation and media silence wsws.org/en/articles/20…

⚠️ SARS-CoV-2 (the virus causing COVID-19) is not classified as a classic oncovirus like HPV, EBV, or HBV, which directly cause ~15% of cancers through well-established mechanisms. However, extensive reviews and studies (including molecular, in vitro, observational, and epidemiological data up to 2026) indicate that SARS-CoV-2 infection can **elevate cancer risk or accelerate progression** in susceptible individuals, particularly through long-term effects like Long COVID, chronic inflammation, and immune changes. Key supporting evidence includes: - Reviews synthesizing molecular pathways (e.g., Ogarek et al., 2023 in *Frontiers in Molecular Biosciences*; Tyagi et al., 2025 in *Biochimica et Biophysica Acta*; Jaiswal et al., 2024). - In vitro studies showing direct effects on cancer cell lines (breast, colorectal, prostate). - Epidemiological signals, such as significantly elevated risks of HPV-related cancers (cervical +67%, anal +92%, etc.) post-infection in large matched cohorts. - Observational data on rapid cancer progression, recurrence, or reactivation of dormant tumors after infection. - Mechanisms overlapping with known oncogenic processes (inflammation, hypoxia, oxidative stress). **Note**: These are primarily mechanistic/hypothetical or associative findings (not proven direct causation in large randomized studies). Some reports note potential oncolytic (anti-cancer) effects in rare cases, but the net evidence leans toward increased risk, especially for lung, colorectal, and virus-associated cancers due to high ACE2/TMPRSS2 expression. Antiviral treatments or vaccines are not the focus here—the query concerns the virus itself. Here are the **5 primary ways** SARS-CoV-2 increases cancer chances, synthesized from all major studies/reviews examined: 1. **Chronic inflammation and cytokine storm** SARS-CoV-2 triggers massive release of pro-inflammatory cytokines (IL-6, TNF-α, IL-1β, IFN-γ, VEGF, etc.), creating a persistent “cytokine storm” and low-grade chronic inflammation. This activates NF-κB and IL-6/JAK/STAT3 pathways, promoting DNA damage, angiogenesis, cell survival/proliferation, invasion, metastasis, and a tumor-favorable microenvironment (e.g., shifting macrophages to pro-tumor M2 phenotype). It also reawakens dormant cancer cells via inflammatory signals. **Evidence**: Central mechanism in nearly every review; directly linked to tumor progression in lung/colorectal cancers; cytokine levels mirror those driving oncogenesis. 2. **Immune dysregulation, lymphopenia, and impaired surveillance** The virus causes profound lymphopenia (depletion of CD4+/CD8+ T cells and NK cells), T-cell exhaustion, overexpression of inhibitory receptors (e.g., NKG2A), and suppression of type I interferons. This weakens anti-tumor immunity, allowing pre-cancerous or dormant cells to escape detection, proliferate, and metastasize. It also disrupts dendritic cells, neutrophils (NETs), and overall adaptive responses. **Evidence**: Documented in severe COVID-19 and post-infection; explains reactivation of latent tumors and synergy with oncogenic viruses (e.g., higher HPV-related carcinoma in situ/cancer rates); parallels cancer immune evasion. 3. **Oxidative stress and genomic instability** Infection downregulates ACE2, leading to angiotensin II accumulation, reactive oxygen species (ROS) overproduction, and oxidative damage to DNA, proteins, and lipids. This causes mutations, epigenetic changes (e.g., altered DNA methylation, miRNA dysregulation, HDAC interactions), and impaired DNA repair—hallmarks of carcinogenesis. **Evidence**: Repeatedly highlighted; ROS links directly to cancer initiation/progression; observed in COVID-19 survivors and tied to genomic instability in multiple pathways. 1/2

Huge new study finding from COVID. 20 months after infection “In cardiovascular disease patients, T lymphocytes remained 72.9% below baseline for 20 months post-infection.” That’s absolutely insane. A 73% persistent drop in the body’s ability to fight infections. This includes your ability to fight cancer…

New study: New evidence for immune deficiency after COVID Infection even 20 months post-infection. Men affected more than women. "Our findings redefine SARS-CoV-2 infection as a condition of long-lasting immune compromise." sciencedirect.com/science/articl…

@usatodaylife This is FALSE information about how HMPV spreads. According to the WHO it is AIRBORNE. Washing your hands will get you nice clean hands. To prevent inhaling a virus, wear a respirator-grade mask (N95, KN95, KF94) in public and in high-risk settings like travel and healthcare.

While there is no specific treatment or vaccine for HMPV, there are steps you can take to help prevent infection. usatoday.com/story/life/hea…

Hospitals stopped masking during an airborne pandemic, and delays in patient admissions exploded.