Dr M.AL-gradey

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@DrsansariOrd ✅ Bacteria ➡️Clostridium tetani ✅Important characteristics ➡️Gram-positive rod ➡️Anaerobic ➡️Spore-forming bacterium ➡️Produces tetanospasmin toxin 🟢The toxin blocks inhibitory neurotransmitters (GABA and glycine) causing muscle rigidity and spasms.

Answer Please ❓

Dx: Erb’s palsy A newborn with a weak or absent Moro reflex on one side after a difficult delivery suggests injury to the brachial plexus (C5–C6 roots), commonly caused by shoulder dystocia or traction during birth.

@kh505043 Dx: Drug Body Packing Clinical presentation May be: ➡️Asymptomatic ➡️Intestinal obstruction ➡️Drug toxicity if a packet ruptures

Spot Diagnosis??? .

@ajlkfad2333 ✅ B) Kinky, brittle hair 🟢features of Menkes disease: •Defect in copper transport Leads to copper deficiency ➡️Characteristic: Kinky/coarse brittle hair Hypotonia Developmental delay Seizures Failure to thrive ➡️The hair is often described as: “Steel wool hair” Pili torti

Q//Which finding is classically associated with Menkes disease? A) Kayser–Fleischer rings B) Kinky, brittle hair C) Café-au-lait spots D) Macroglossia

@abdulra_hman11 ✅ داء كرون (Crohn’s disease) لماذا يسمى Cobblestone؟ لأن المخاطية تصبح: فيها تقرحات طولية وعرضية مع مناطق مخاطية متورمة بينها فتعطي شكلاً يشبه الحجارة المرصوفة (cobblestones).

تخيل ان هذا منظار للأمعاء هذا المنظر اسمه cobblestone appearance في اي حالة نجد هذا المنظر ؟

@modernHealthMe Noma (also called Cancrum oris) is a rapidly progressive gangrenous infection of the mouth and face, mainly affecting malnourished children in poor-resource settings. Begins as gingivitis or oral ulcer

What's the diagnosis??

@DrsansariOrd ✅ A. Erb’s palsy A newborn with a weak or absent Moro reflex on one side after a difficult delivery suggests injury to the brachial plexus (C5–C6 roots), commonly caused by shoulder dystocia or traction during birth.

A newborn with weak Moro reflex on one side after difficult delivery may have❓ A. Erb’s palsy B. Hydrocephalus C. Neonatal sepsis D. Hypoglycemia

Two identical twin doctors joined the same hospital on the same day. Both wear the same scrubs, same watch, same hairstyle, even the same perfume. 😭 One is a radiologist. The other is a gynaecologist. The nurses say there’s only ONE tiny detail that exposes the gynaecologist twin… 👀 Can you spot which doctor is the gynaecologist?

🔴Ameloblastoma is a benign odontogenic tumor that originates from the enamel-forming cells of the jaw. ➡️Robinson's Definition: 🔴Ameloblastoma is usually Unicentric, Nonfunctional," Intermittent in growth, Anatomically benign, and Clinically persistent. (Mnemonic: UNIAC)

@IhabFathiSulima Ameloblastoma is a benign odontogenic tumor that originates from the enamel-forming cells of the jaw. Robinson's Definition: ➡️Ameloblastoma is usually Unicentric, Nonfunctional," Intermittent in growth, Anatomically benign, and ".Clinically persistent (Mnemonic: UNIAC)

What is the diagnosis ⁉️

@IhabFathiSulima Dx:Superior Vena Cava Obstruction ➡️Common causes include lung cancer, lymphoma, and thrombosis from central venous catheters. ➡️Patients present with facial swelling, distended neck veins, dyspnea, cough, and upper limb edema.

What is the description or diagnosis?

@docakx This is sometimes called aleukaemic leukaemia. Bone marrow infiltration and systemic symptoms occur later. Sometimes myeloid leukaemia relapses in the skin after apparently successful treatment of bone marrow. It is then called ‘extramedullary’ leukaemia.

@docakx Leukemia cutis occurs when malignant white blood cells infiltrate the skin. It typically presents as firm,painless,and rubbery bumps,flat patches,or thickened plaques. The skin lesion is the very first sign of the haematological malignancy in 7% of patients with leukaemia cutis.

Patient of Acute Myeloid Leukemia. What is this?

@docakx What type of rare cancer is it?

Tulsi Gabbard's husband Abraham diagnosed with an extremely rare form of bone cancer. She announced her resignation to tend to him.

@ajlkfad2333 @IhabFathiSulima @docakx Diagnosis here is Hemorrhagic Stroke

Answer: Hemorrhagic brain tumor (most likely Glioblastoma) ➜ Key imaging feature: Heterogeneous lesion with mixed density → hemorrhage within tumor. ➜ Edema pattern: Marked perilesional edema → suggests chronic neoplastic process, not acute bleed. ➜ Mass effect: Significant midline shift and compression → expansive space-occupying lesion. ➜ Morphology: Irregular infiltrative margins → consistent with high-grade glioma (GBM).

Came in for stroke like symptoms. What's the diagnosis?🤔 @IhabFathiSulima

@DrsansariOrd @IhabFathiSulima @docakx Exactly, hemorrhagic stroke Thank you so much for your detailed information

This is far more than just “stroke-like symptoms” — this is a large acute lobar intracerebral hemorrhage with dangerous mass effect. A few additional high-yield points: 🚨 Why this is immediately life-threatening The major killer here is not only the bleed itself, but: Rapid rise in intracranial pressure (ICP) Progressive midline shift Risk of subfalcine or transtentorial herniation The compressed ventricle and distorted anatomy already suggest severe intracranial hypertension. 🧠 Important localization clue Because the hemorrhage is: Lobar Cortical/subcortical Not centered in basal ganglia …it becomes less typical for chronic hypertension alone. That is why: CAA becomes highly likely in elderly patients AVM/tumor/cavernoma must be excluded in younger patients 📌 CT appearance pearl Acute blood appears hyperdense (bright white) on non-contrast CT because freshly clotted blood has high protein and iron concentration. ⚡ “Stroke mimic” reminder Not all sudden focal deficits are ischemic strokes. A non-contrast CT is critical before thrombolysis because giving tPA to this patient would be catastrophic. 🚑 Neurosurgical red flags present here Large hematoma volume Midline shift Mass effect Cortical superficial location These are exactly the features that trigger urgent neurosurgical consultation. 🧠 Classic teaching: > “Deep hemorrhage = hypertension until proven otherwise. Lobar hemorrhage = think beyond hypertension.”

This is far more than just “stroke-like symptoms” — this is a large acute lobar intracerebral hemorrhage with dangerous mass effect. A few additional high-yield points: 🚨 Why this is immediately life-threatening The major killer here is not only the bleed itself, but: Rapid rise in intracranial pressure (ICP) Progressive midline shift Risk of subfalcine or transtentorial herniation The compressed ventricle and distorted anatomy already suggest severe intracranial hypertension. 🧠 Important localization clue Because the hemorrhage is: Lobar Cortical/subcortical Not centered in basal ganglia …it becomes less typical for chronic hypertension alone. That is why: CAA becomes highly likely in elderly patients AVM/tumor/cavernoma must be excluded in younger patients 📌 CT appearance pearl Acute blood appears hyperdense (bright white) on non-contrast CT because freshly clotted blood has high protein and iron concentration. ⚡ “Stroke mimic” reminder Not all sudden focal deficits are ischemic strokes. A non-contrast CT is critical before thrombolysis because giving tPA to this patient would be catastrophic. 🚑 Neurosurgical red flags present here Large hematoma volume Midline shift Mass effect Cortical superficial location These are exactly the features that trigger urgent neurosurgical consultation. 🧠 Classic teaching: > “Deep hemorrhage = hypertension until proven otherwise. Lobar hemorrhage = think beyond hypertension.”

@IhabFathiSulima Often associated with pulmonary contusion (bruising of the lung), which further compromises respiratory function.

@IhabFathiSulima ✅Pathophysiology: The flail segment moves paradoxically during respiration: 🔸Inspiration: flail segment moves inward (opposite to normal chest wall movement). 🔸Expiration: flail segment moves outward. This paradoxical movement impairs ventilation and oxygenation.

What is the diagnosis?