Philip J Podrid MD

859 posts

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Philip J Podrid MD

Philip J Podrid MD

@PPodrid

ECG Educator | Electrophysiologist | Professor @The_BMC | Alumni @BrighamFellows & @NYUMed | All tweets are my personal opinion 🩺

Boston, MA Katılım Aralık 2019
53 Takip Edilen5.6K Takipçiler
Philip J Podrid MD
Philip J Podrid MD@PPodrid·
Atrial flutter with variable av block(2:1 and 4:1). Atrial rate is reg at about 350 and whenever atrial rate is reg and >320 this is termed atypical atrial flutter. Typical atrial flutter has a reg atrial rate of 260-320 although may be slower due to aad or disease of la.
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Philip J Podrid MD
Philip J Podrid MD@PPodrid·
@ecgandrhythmRoe NSR with 2:1 AV block due to Mobitz 2 as with 1:1 conduction the PR intervals are the same. Also ventriculophasic arrhythmia as pp intervals around QRS complex are shorter than the pp intervals without QRS complex.
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Dr. Andreas Roeschl
Dr. Andreas Roeschl@ecgandrhythmRoe·
A 65-year-old woman with this ECG. What would you do if the patient were experiencing symptoms (syncope) and if she were not?
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Philip J Podrid MD
Philip J Podrid MD@PPodrid·
RV pacemaker that is A sensed V paced. Sinus rate is 120 bpm. Pauses due to non sensed p wave and hence no ventricular paced complex. This is because the sinus rate slightly increased and the p wave was within the pvarp. Hence the upper rate limit of the pacer is about 120bpm.
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Philip J Podrid MD
Philip J Podrid MD@PPodrid·
Regular narrow complex tachy at a rate of 270 bpm-hence atrial flutter with 1:1 conduction.ST-TW changes likely due to superimposed flutter waves. Also seen is electrical or QRS alternans (v3-v5).
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Philip J Podrid MD
Philip J Podrid MD@PPodrid·
Underlying rhythm is AF. Biventricular pacemaker A sensed V paced and tracking AF at its upper rate limit of about 120 bpm.
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Philip J Podrid MD
Philip J Podrid MD@PPodrid·
Rhythm is irregularly irregular. Atrial waveforms in lead v1 very prominent but they are irreg in amplitude, interval and morphology. Hence not a flutter where atrial waves are uniform. This is coarse AF. There is also LVH with associated ST-T wave changes.
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Philip J Podrid MD
Philip J Podrid MD@PPodrid·
Patient with no heart disease presents with new onset of an irregular and fast heart rate. What is the rhythm?
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Philip J Podrid MD
Philip J Podrid MD@PPodrid·
@Frances98392343 @syamkumarmd @MethaqOrman @MaruanCarlos @The_Nanashi_O @ecgrhythms @UlhasDr @DidlakeDW @DrRajeshG1 @AslangerE @adribaran @BrooksWalsh @doctor_roig @KostekMilan Bundle conduction is all or none. Incomplete bundles don’t exist. In this case, the small r’ in v1 is an ivcd to the rv and has been termed a crista pattern as the last part of the rv to be depolarized is the crista supraventricularis and conduction to this structure is delayed
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Philip J Podrid MD
Philip J Podrid MD@PPodrid·
NSR with pvc and then two APCs followed by a long RP tachy that terminates abruptly to NSR. Terminates without a p wave-ie last complex before it end does not have a p wave after it. This is the wss say atrial arrhythmias terminate. Hence this is an atrial tachycardia.
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Philip J Podrid MD
Philip J Podrid MD@PPodrid·
37 year old woman with a history of palpitations. An ecg was obtained during an episode. What does the ecg show and what is the etiology of the palpitations? #PodridECGs #ECG # needed #cardiotwitter
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Philip J Podrid MD
Philip J Podrid MD@PPodrid·
NSR with RBBB and lafb- termed bifascicular disease. The pauses are the result of non conducted PACs occurring in a trigeminal pattern.
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Philip J Podrid MD
Philip J Podrid MD@PPodrid·
Sinus rhythm with polymorphic VT. Sinus complex has very prolonged QT interval of 0.60 sec. Hence this is called torsade des pointes. It may be drug induced or congenital. This is establish by history of any current drug use. Given multiple episodes over time likely congenital.
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Philip J Podrid MD
Philip J Podrid MD@PPodrid·
NSR with one pac. Tall r wave in v1 as well as right axis and p-pulmonale (Himalayan P wave)-hence rvh. Deep S wave in v2 and tall r wave v4 c/w LVH. Hence biventricular hypertrophy with assoc state changes.
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