don_x

@BerbarianWizard Wouldn’t low dose DHT, even in the form of a gel, cause some level of systemic suppression? Or would this simply correct itself after stopping use?

Best option: get testosterone dialed naturally. Testosterone is an output signal. You restore energy production and regulatory inputs, and testosterone follows. Main drivers: thyroid function, total calories, carbohydrate availability, micronutrients (zinc, selenium, vitamin A …etc.), sleep and circadian rhythm, stress load, inflammation, liver function, insulin sensitivity, body composition, training load and recovery. Second: thyroid hormones. They increase mitochondrial output, cholesterol utilization, steroidogenesis, and testicular responsiveness to LH. Hypothyroid states reduce androgen production efficiency. Thyroid alone does nothing if energy intake, stress, and inflammation are not corrected. Third: short-term low-dose DHT. It increases androgen receptor activation and opposes estrogenic signaling. This can shift steroid metabolism toward 5α-reduction and may upregulate 5α-reductase activity, creating a positive feedback that favors a higher androgen-to-estrogen ratio. The effect persists only if upstream drivers are corrected. Estrogen stimulates cortisol, cortisol increases aromatase expression, and aromatase increases estrogen, maintaining a reinforcing loop. Worst: TRT. It fixes the output without restoring the system. In an unhealthy metabolic state, testosterone becomes substrate for aromatization, increasing estrogen and creating additional problems that often require further interventions. It turns into a management loop rather than correcting the root cause. Second worst: HCG. It replaces LH at the testicular level. Chronic use suppresses endogenous LH production and recovery can be slow after discontinuation. It commonly increases intratesticular aromatase and estrogen. Enclomiphene and other SERMs: they increase LH by blocking estrogen feedback at the hypothalamus and pituitary, but they are not neutral. They act as antagonists centrally and partial agonists in other tissues depending on receptor subtype. This can lead to visual disturbances, mood instability, and unfavorable lipid changes. They artificially stimulate LH while leaving the underlying metabolic and endocrine context unchanged, so the system remains dependent on continued use.

@FefeDemeny COME ON FEFE, DAY ONE SUPPORTER HERE

I'M LIVE GIVING AWAY $500.000 WORTH OF STUFF WANT A CHRISTMAS PRESENT!? COMMENT BELOW 👇 youtube.com/watch?v=K47nr0…

@BigCheds THATS MY QUANT!!

8 years ago. Now celebrating 7+ years of being cancer free 🤝

@FefeDemeny He defo trolling Fefe😂

When you think you’ve seen it all…

@BigCheds Cheds we need your Quant skills right now

Deep in the money

$TRUMP coin major bounce into underside of lost support

@BigCheds Capo is no longer my quant

Capo is eating ramen tonight

@BigCheds Capo is now my quant

Capo is eating filet mignon tonight