Joyce Bwombengi

The draft of the @goKDIGO 2026 Clinical Practice Guideline for Acute Kidney Injury (AKI) and Acute Kidney Disease (AKD) is now available for public review through 🗓️ April 27, 2026 #ISNWCN 🇯🇵 #WCN26 🇯🇵 #WCN2026 🇯🇵 #Nephpearls You can download the guideline draft and submit comments via the feedback form 👉 kdigo.org/guidelines/acu… 👉 FEEDBACK Form surveymonkey.com/r/GTGNFHG

👍Approach to Nephrotic syndrome based on anti nephrin antibody🆕 @NDTsocial @hjanders_hans doi.org/10.1093/ndt/gf…

PIGN that didn't read the textbook. Young adult with nephritic syndrome + recent step throat. +ASO. Low C3/C4. UA with 2+ blood and 1+ protein. DPGN with full house staining and massive subendo deposits. Serologic w/u (-). Resolved with supportive care. #renapath #nephrology

🫘Nephrotic 🆚Nephritic syndrome @thecurbsiders

The endothelium response to kidney injury dlvr.it/TRgL0H

🔥Hyponatremia made simple Dr Ajay Kher: 1️⃣ Serum Osm → hypo vs pseudo 2️⃣ Urine Osm → <100 = polydipsia, >100 = ADH present 3️⃣ Urine Na → <20 = volume depletion, >40 = SIADH 🙇‍♀️Structured thinking = faster diagnosis. #CNSICON2026 @CNSI2026 @VinBhargava @rajdebsaha @drvt87

🧬Semaglutide in CKD—mechanistic depth • ↓ intrarenal fat • ↑ glomerular hemodynamics • ↓ inflammation & fibrotic signaling • Endothelial reprogramming → ↓ albuminuria + preserved GFR @KatherineTuttl8 @isn_india @ISNeducation @cnsi2026 @Vinbhargava #CNSICON2026

⚔️ CRRT vs SLED—evidence meets real-world practice Dr. Aakash Singada makes a strong case for SLED: 📊 Comparable outcomes to CRRT 🏥 Better feasibility in resource-limited settings 💰 Cost-effective 🔌 Easier access & mobility @aakashshingada @rajdebsaha #CNSICON2026

Hemodialysis Facilities, Vascular Access Surgeons, and Central Venous Catheter Use: A Retrospective Cohort Study bit.ly/3ZK5TWp #OpenAccess @MKOerline @RichardHirth @dorkstweet @UMichKidney

En biopsias del trasplante renal hechas bajo indicación clínica, desmopresina IV 0.3 μg/kg profiláctica, comparado a placebo; no evitó los sangrados mayores, ni menores Ensayo clínico BRIDGE CKJ 2026 academic.oup.com/ckj/advance-ar…

Delighted that FIND CKD has meet its primary endpoint. Fantastic news for patients with non-diabetic CKD, including those with GNs, including IgAN Thank you to Bayer, all the investigators, study teams & patients who have made FIND-CKD possible bayer.com/media/en-us/fi…

Ten tips on how to prevent and manage post-transplant lymphoproliferative disease in kidney transplant recipients doi.org/10.1093/ckj/sf…

MINI REVIEW: A Practical Primer on How to Detect and Treat Depression in CKD bit.ly/4agFzJU (FREE) @LParkerGregg1

@BrownHospMed @Radiopaedia Nutcracker syndrome(renal vein entrapment syndrome)-a vein compression disorder.It occurs when arteries,most often the abdomen’s aorta and superior mesenteric artery,squeeze the left renal(kidney)vein.Dx-CT,DUS,venographyTx-Open surgery for posterior nutcracker syn.

#POCUS answer: 1. SMA 2. Left renal vein. Only 36% got it right. This is a classic image illustrating nutcracker syndrome as some of the friends mentioned in the thread (See images 👇) compression ratio = anteroposterior diameter of the pre-compressed vein divided by that of the compressed vein. Ratio > 2.25 has high sensitivity and specificity for this syndrome.

If you are not familiar with nutcracker syndrome, it refers to compression of the left renal vein between the aorta and proximal superior mesenteric artery. Can cause microscopic/gross hematuria +/- flank pain, orthostatic proteinuria. #MedEd #Nephrology

Nutcracker syndrome #POCUS #IMPOCUS #MedEd

Autosomal dominant polycystic kidney disease (ADPKD) is the most common genetic cause of chronic kidney disease. A new Review focuses on current approaches to diagnosis, treatment, and specific aspects of clinical management. Read more: spkl.io/6016AxPWO

📢NEW pro / con debate: Simultaneous or sequential treatment of IgA nephropathy - specific disease drivers and non-specific consequences of nephron loss? 📖doi.org/10.1093/ckj/sf…

🌞 Malar Rash: Beyond Lupus A malar rash is an erythematous rash over the cheeks and bridge of the nose, classically resembling a butterfly. Not every malar rash is lupus Here are a few differentials of malar rash👇👇 🧠 Rheumatological Conditions 🛡️ Systemic Lupus Erythematosus (SLE) •Fixed erythema over cheeks and nasal bridge •Spares nasolabial folds •Photosensitive 💜 Dermatomyositis •Does NOT spare nasolabial folds •Heliotrope rash •Gottron’s papules •Proximal muscle weakness 🌿 Dermatological Conditions 🌸 Rosacea •Central facial erythema •Telangiectasia •Papules and pustules •Does not spare nasolabial folds 🧴 Seborrheic Dermatitis •Prominent involvement of nasolabial folds •Greasy yellow scales ☀️ Photodermatitis •Sun-exposed distribution •Clear photosensitivity history 🧪 Contact Dermatitis •Pruritic eruption •Exposure related 🦠 Infectious Causes 🔥 Erysipelas •Acute onset •Tender, well-demarcated erythema •Fever and systemic symptoms 🧫 Parvovirus B19 •“Slapped cheek” appearance •More common in children •Often associated with viral prodrome #RheumattDoc #MedTwitter #RheumTwitter #Medicine #rheumatology @DrAkhilX @IhabFathiSulima @CelestinoGutirr @DurgaPrasannaM1