Matt Wilson

19.4K posts

Matt Wilson

Matt Wilson

@stringerbell99

Entrou em Ocak 2010
612 Seguindo198 Seguidores
Matt Wilson
Matt Wilson@stringerbell99·
I shouldn’t have to make it at all. You just refuse to recognize something that’s inconvenient to you, and it seems there’s no end to how far you will go to continue denying it. I’m simply demonstrating that you DO recognize there are risk factors for heart disease, even though they don’t meet the standard of evidence you claim LDL needs either. Either LDL is a risk factor, or there’s another are no known risk factors. Which is it?
English
0
1
0
14
Roxana Soetebeer, MPHC NNP MHP PFC
Roxana Soetebeer, MPHC NNP MHP PFC@REV_Insulin_Res·
Oh boy, you still cannot navigate observational studies. I am not a doctor, yet I understand that observational studies, no matter how much you like the results, can never establish causality. Yep, never ever. Sorry about that.
Dr Alo, DO, FACC@MohammedAlo

Does eating less saturated fat improve mortality and save lives? #saturatedfats #cholesterol #mortality #heartdisease #cardiology #doctorsoftiktok #medicine #CapCut #CardioTwitter #MedTwitter

English
11
3
33
6.4K
Matt Wilson
Matt Wilson@stringerbell99·
@REV_Insulin_Res @RobertM40897248 I don’t want anything. Just pointing out the inconsistency in your logic, and yes, you did call them risk factors, which they are. As is LDL/apob.
English
1
0
0
15
Matt Wilson
Matt Wilson@stringerbell99·
Here you are saying gum disease is a risk factor for CVD, and there’s another with you saying inflammation is a primary driver of CVD. So my question is still, why do you hold LDL to a different standard? I already know the answer, because I used to do the same thing, but it’s something you should realize yourself. x.com/rev_insulin_re…
English
1
0
0
17
Matt Wilson
Matt Wilson@stringerbell99·
And that’s not a good characterization of Mendelian randomization studies. They use genetic SNP’s that are strongly correlated with high or low lifetime cholesterol levels with hundreds of thousands of data points as a proxy for lifetime levels. This cancels out a lot of the noise you see in observational studies like reverse causation and gives a clear picture of the increased risk.
English
1
0
0
8
Roxana Soetebeer, MPHC NNP MHP PFC
Roxana Soetebeer, MPHC NNP MHP PFC@REV_Insulin_Res·
You mean when they study gene snippets and don't even bother to measure LDL? Please understand, I am not saying LDL doesn't matter. I am saying science cannot establish a causal relationship between LDL and heart disease. I agree that we cannot do lifelong RCTs. But not being able to do that does not mean we can settle for lower evidence.
English
2
0
0
23
Matt Wilson
Matt Wilson@stringerbell99·
There’s no way to do a lifelong randomized controlled trial with 10,000s of thousands of people, but we can do Mendelian randomization studies that compare genetic mutations that cause lifelong high or low LDL. They show a clear linear risk between lifelong LDL exposure and cardiovascular mortality. It’s a key piece of evidence that’s also confirmed by observational data and randomized controlled trials. I guess according to your logic there are no known risk factors for heart disease? Or do you hold LDL to a different standard than other risk factors because it’s more convenient to your beliefs?
English
1
0
0
13
Matt Wilson
Matt Wilson@stringerbell99·
@millepun @ifixhearts God didn’t create us to be diabetic or obese or a smoker, etc either. These are some of the other risk factors for heart disease.
English
0
0
0
20
M for Electric ⚡️🔋
M for Electric ⚡️🔋@millepun·
@stringerbell99 @ifixhearts Many people doing healthy diet and life style, great metabloic health and low imflammatory marker. But still have high LDL. If LDL turely an isolated risk factor, and still require a human-made drug to low it, then God didnt make human right.
English
2
0
0
14
Heart Surgeon Dr. Philip Ovadia
Heart Surgeon Dr. Philip Ovadia@ifixhearts·
Why do I end up doing heart surgery on so many patients who have a normal cholesterol level and who aren't obviously obese? That question led me to go back to the basic science and to understand the importance of metabolic health.
English
16
16
184
14.2K
Matt Wilson
Matt Wilson@stringerbell99·
@millepun @ifixhearts No, what’s wrong is our modern way of life. The modern hunter gather groups that still live a traditional lifestyle generally have LDL in the 50-85mg/dl range and very rarely develop heart disease. All other primates are in this range as well.
English
1
0
0
24
Matt Wilson
Matt Wilson@stringerbell99·
@REV_Insulin_Res @RobertM40897248 That’s well established by other literature, but there are many other risk factors as well. Whatever your risk level, high LDL only ever raises your risk.
English
1
0
0
21
Roxana Soetebeer, MPHC NNP MHP PFC
Roxana Soetebeer, MPHC NNP MHP PFC@REV_Insulin_Res·
@stringerbell99 @RobertM40897248 I’ll admit, finding populations with low LDL without statins is surprising. They don’t represent all non-modern groups, but still. That said, this is observational data. It does not establish causality between LDL levels and heart disease.
English
1
0
1
98
Ken D Berry MD
Ken D Berry MD@KenDBerryMD·
If there was even a shred of curiosity or academic integrity in the nutrition research space, researchers would be falling over themselves to study this self-selected group of carnivores! Why don't they develop scurvy? Why isn't there a heart disease spike in Carnivores? Shouldn't they develop Type 2 Diabetes at higher rates? Colon cancer rate hockey-stick? Rampant inflammation (measurable) ? Pounds of meat rotting in their colon? Nothing?? Remember, "they" keep telling us that one extra egg/day increases risk. 2 ounces of red meat increases risk. CARNIVORES EAT 2 POUNDS OF MEAT AND A DOZEN EGGS/DAY! Shouldn't the signal be off the charts??
Tennessee, USA 🇺🇸 English
102
293
1.8K
44K

Descobrir

@RobertM40897248 @REV_Insulin_Res @elonmusk @BarackObama @taylorswift13 @cristiano @BillGates @NASA