FatLiverDoc

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FatLiverDoc

FatLiverDoc

@LiverFat

Liver doctor specializing in the treatment of fatty liver disease and obesity.

가입일 Şubat 2020
53 팔로잉73 팔로워
FatLiverDoc
FatLiverDoc@LiverFat·
@liverUSA So very sad for his family, such a terrible loss for the Chicago liver family and the hepatology community as well.
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American Liver Foundation National
American Liver Foundation mourns the tragic loss of Josh Levitsky, MD, MS, FAST, FAASLD, a highly respected, devoted and patient-centric leader in the field of liver transplantation. liver.news/JLevitsky
American Liver Foundation National tweet media
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FatLiverDoc
FatLiverDoc@LiverFat·
All’s good at the @AlfDebates . Great debate on whether a mandatory lifestyle intervention should be instituted for patients being transplanted for NASH. Important and nuanced topic!
FatLiverDoc tweet media
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FatLiverDoc
FatLiverDoc@LiverFat·
@gscottwinder I haven’t much time researching the actual assay to see how the values are calculated, but I believe there is a rough correlation although I am not sure it is a linear one. I say this based on my loose correlations between with level of reported etOH intake and PeTh level.
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FatLiverDoc
FatLiverDoc@LiverFat·
@gscottwinder Then I interpret the test like I was still a lab scientist... interpret the number in context, not the lab given cutoffs.
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FatLiverDoc
FatLiverDoc@LiverFat·
@gscottwinder I tell people the test I am running is not supposed to be positive with a single slip but rather from repeated chronic significant alcohol use. This is a way for them to know if the amount they are exposed to if any is too much. But tell them I believe them if they say it’s none.
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FatLiverDoc 리트윗함
Michael Albert, MD
Michael Albert, MD@MichaelAlbertMD·
Why therapeutic low-carb + HP is so effective in T2DM: Graph A: washout diet vs ADA diet (Carb 55%, Protein 15%, Fat 30%) Graph B: washout diet vs therapeutic low-carb + HP (Carb 20%, Protein 30%, Fat 50%) *Glycemic control is superior with low-carb (non-keto) + high protein*
Michael Albert, MD tweet media
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FatLiverDoc
FatLiverDoc@LiverFat·
FatLiverDoc@LiverFat

@GiJournal @AtoosaRabiee @mqkMD @ijlalakbar @SultanMahmoodMD @DrMohdZein @GI_Pearls @LiverToronto @NAFLDSupport @LizzieAbyMD @stevenbollipo Though it predicts HRS development, does diminished cardiac reserve alone justify prioritization for transplant if diminished cardiac reserve does not predict transplant-free survival and did not impact survival in those that did not develop HRS?

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GI Journal Club
GI Journal Club@GiJournal·
Its time for another exciting JC discussion. Here is a quick summary of the article
GI Journal Club tweet media
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FatLiverDoc
FatLiverDoc@LiverFat·
@AtoosaRabiee @SultanMahmoodMD @GiJournal @MaureenWhitsett @liverazzi @ebtapper @ArpanPatelMD @JBenhammouMD @BloomPringle @JonathanStineMD @gnioannou @drkeithsiau @Will_Sanchez_MD @BWSadowskiMD @SanchezLunaMD @AhmadBazarbashi agreed, I think that once you have HRS and are likely on chronic midodrine therapy, NSBB are out. but agreed, maybe this is another clinical characteristic (with SBP, refractory ascites) that should lead to hold BB. but again, need mortality data.
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FatLiverDoc
FatLiverDoc@LiverFat·
@AtoosaRabiee agreed, but in the end, this study just shows that as they get sicker, they are more likely to get cirrhotic cardiomyopathy. The "new" (quotes intentional) thing was that cirrhotic cardiomyopathy predicts HRS. but like you said.. does it change ur mgmt.
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