JM&Co

When you are dealing with chronic lyme, it is rarely just one thing. These stealth pathogens like to travel in packs. Finding the root cause means acknowledging that coinfections like babesia, bartonella, and anaplasma often complicate the picture. It is a lot to manage, but understanding the full toxic load is the only way to move toward real lyme recovery. Don't let the complexity overwhelm you. Focus on steady, consistent support for your immune dysfunction. You have to look at the whole system to get your life back.

LYME disease/ The Dental Connection A 67-year-old “HEALTHY/ SAFE & EFFECTIVE” root canal aka DEAD tooth Tooth roots SHOULD NOT be DARK! The following/ highlighted microbes were detected in the submitted sample:- B. burgdorferi Osp A-NPS B. burgdorferi Osp B-NPS Babesia duncani-NPS Borrelia recurrentis-NPS The following are the “HEALTH BENEFITS” of the:- B. burgdorferi Osp A-NPS Contributing to the pathology of LYME disease B. burgdorferi Osp B-NPS There are no known health benefits of Borrelia burgdorferi Outer Surface Protein B (OspB) for humans. OspB is a virulence factor produced by the spirochete that causes LYME disease, and it contributes to the pathogenesis of the infection rather than providing therapeutic value. Babesia duncani-NPS The parasite Babesia duncani itself causes babesiosis. It infects red blood cells and can lead to severe symptoms such as fever, chills, sweats, and potentially life-threatening complications like respiratory distress or organ failure, particularly in immunocompromised individuals. Borrelia recurrentis-NPS Borrelia recurrentis has no health benefits; it is a pathogenic bacterium and the sole causative agent of louse-borne relapsing fever (LBRF), a serious infectious disease. Pathogenicity: B. recurrentis causes systemic inflammation, recurrent high fevers, and severe complications such as acute respiratory distress syndrome (ARDS), myocarditis, hepatitis, and neurological issues. Mortality: Without treatment, mortality rates can exceed 30% to 80% during epidemics, though this drops to less than 5% with appropriate antibiotic therapy. Immune Evasion: The bacterium employs sophisticated immune evasion strategies, including antigenic variation of surface proteins and the binding of host complement regulators (such as Factor H and C4BP) to survive in human blood and tissues. Know the RISKS BEFORE consenting to a root canal. Slide credit/ Dr. Kelly J. Blodgett DMD NMD