Kari Tuominen

JADEITES LIVE🎙️ Big reveal incoming... Next week we showcase a core component of the Jade City Protocol. Be first to see it 👉 x.com/i/spaces/1Mnxn… 📅 23rd Sept 3PM UTC 🏆 3 x $100 in $JCT to win ✨ Like & Share to enter #RWA

Xandeum isn't about replacing what exists, it's about unlocking what hasn’t been built yet. We need more than speed, we need a system to store ideas.

Solana validators process thousands of transactions per second, there’s no time to fetch data from disk. We’re building the storage layer that smart contracts ACTUALLY need.

Running a pNode on Xandeum doesn’t require a server farm or a PhD. Plug in, help power web3, and earn while you’re at it.

Web3 doesn't feel quite right 😖 We’re building for a future where dApps are dynamic, data-rich, and truly alive.

Decentralized storage isn’t optional, it’s got to happen. We can’t call it web3 if our data still lives in web2 silos.

Solana devs store everything in RAM (Accounts). This isn't scalable, but Xandeum is adding a storage tier that feels native, but scales like the cloud.

pNode Dutch Auction launches Sept 6: 49 Deep South Era nodes with NFT multipliers (up to 11x) to boost STOINC. Munich release live—upgrade your pNode now for devnet launch at 150 nodes. Herrenberg adds search next. Vote on auction start time via DutchDecider app by Sept 5.

You just hit a 10x on $XAND. What’s the first thing you're storing on your pNode?

Details: xnd.to/AuctionAndSTOI…

@xandeum Xandeum is a solution!

Web3 can’t evolve if every dApp is designed around scarcity. Xandeum is here to flip the model: from scarcity to scale.

If you’ve ever tried to store a meaningful amount of data on-chain, you know the pain. What if it just worked, like every other file system you’ve ever used?

🚨 Roll call! 🥁 Time to update to v0.3.3 – it's super straightforward: SSH into your pNode, hop over to localhost:3000 in your browser, and hit that Update button. No terminals or code required – just one easy click!

Xandeum is building for the apps that haven’t been built yet—for the future Reddit, YouTube, and Wikipedia of Web3. Because today’s storage limitations are blocking tomorrow’s breakthrough ideas.

Web3’s next breakthrough isn’t more chains—it’s scalable storage. 🧑‍💻 Social networks, AI, virtual worlds, and DeFi all need fast, decentralized data access.

The government HAS NO CAPS ON CHOLESTEROL..for ten years now. So why didn’t they tell you? 🤔 The U.S. government quietly dropped its cholesterol cap in 2015 (the American Heart Association did the same in 2013), yet most people still think dietary cholesterol is something to fear. Why? Maybe because our government is too afraid to publicly correct its mistake. There was no information campaign, no big announcement—mostly just silence. Here’s the truth: cholesterol isn’t the enemy. The government even funded many large-scale, randomized clinical trials to test the link between #cholesterol and heart disease. What did they find? 👇 No proof that eating meat, butter, and cheese leads to heart disease. Some studies even showed the opposite: the more people lowered their cholesterol, the more likely they were to die from heart disease. So what happened to those studies? Buried. Suppressed. Ignored. One sat in the basement of the NIH for years, never published. Another delayed publication for 16 years. And while the government dropped its cholesterol caps, it still says that healthy diets are "lower in cholesterol" It's time to ask: why are we still being misled?

🚨Did you know? Some people have reduced their insulin intake by 150 units in just 8 days on a low-carb diet. Carbohydrate restriction isn’t just effective—it’s life-changing. #DiabetesReversal #LowCarbLiving Read the study here: sciencedirect.com/science/articl…

The Elephant in the Room is that the skeletal muscles' fuel supply does not influence its ability to produce work. The muscle does just as well burning fat or carbohydrate. That's what our work over the past few years has shown. The problem is that exercise physiologists - myself very much included - have thought of human exercise performance as if skeletal muscle works like a petrol-driven engine. All of us know that the petrol driven engine requires the provision of more petrol for our cars to travel faster. So we press the accelerator which supplies more fuel to the engine which produces a more powerful combustion which then makes the pistons travel faster which ultimately makes the car's wheels rotate faster. So it's obvious - more fuel = more speed. And then we promote the idea that carbohydrates - muscle glycogen in particular - can produce ATP more rapidly than the slow-burning fats. So if you want to fill up on "jet fuel" you'd better eat more carbs to stock your muscles with the ultimate jet fuel, muscle glycogen. But we conveniently forget that to provide more fuel to our car's engine, our brains must activate the calf muscles of our right legs to press harder on the accelerator. So what determines the car's speed? Answer: How hard the human driver presses on the accelerator. Because no pressure = no speed. Simple. And the human engine is no different. So for human skeletal muscle to burn/use more oxygen/fuel/ATP, it must first activate more skeletal muscle actin/myosin cross bridges which then use the ATP generated by the mitochondria to power a more powerful muscle contraction. The mitochondrial-generated ATP does not drive this process. The process is activated by calcium released as a result of increased brain to spinal cord to peripheral nerve activity. Thus the most powerful/fastest athletes may well be those who can produce ATP at the highest rates. But that's not the immediate cause of their superior ability. Rather it is principally a result of their greater capacity to release the activator calcium which then activates the formation of a greater number of actin-myosin cross bridges. The point is: If you want to know how changing the fuel supply to the muscle will impact the muscle's function, it's simplistic to think in terms of rates of oxygen or ATP provision. The key is how does that change (if at all) the number of actin/myosin cross bridges that the brain is prepared to activate in the exercising muscles. Our data suggests that the only circumstance in which the fuel choice becomes critically important is how it affects brain function. In particular whether the brain senses that the anticipated exercise bout will cause the blood glucose concentration (the small glucose pool) to fall. If the brain anticipates that this will happen, then, well before the blood glucose concentration falls to levels that will threaten brain damage, it will (subconsciously) reduce the number of actin/myosin cross bridges that it will allow to be activated in the exercising skeletal muscles. As a result the athlete will slow down. At the same time the brain generates increased sensations of fatigue to warn the athlete that not everything is OK and he/she needs to slow down to avoid bodily harm. @LoreofRunning1 @PhilipPrins11 @AKoutnik @PaulBLaursen @theplews1 @CarynZinn @sweatscience @brady_h @zoeharcombe @DoctorTro @bigfatsurprise

Web3 history rewards those who get in early. Solana’s biggest validators started small. Now, Xandeum’s pNodes are launching soon, and the opportunity is here.