Trinity

Earlier today I was looking at my 42,000 @AlaskaAir miles and dreaming about where I could go for free at the end of a year. Less than 12 hours later, those miles served a better purpose to help an old classmate attend a funeral this week back home. It wasn’t even a choice.

this is one of my fav the hunger games edits i’ve made

WOW this blew up! I'm glad so many people are loving Tatiana's narration of The Hunger Games trilogy! 🥹 Suzanne Collins, if you ever write another THG prequel book, PLEASE write a character Tatiana can play in the movie adaptation! She would make an INCREDIBLE Head Gamemaker!

Peter Thiel in 2009: "I no longer believe that freedom and democracy are compatible." Palantir is ready to dismantle both. Here's a breakdown of its little manifesto:

A groundbreaking study has pinpointed a microscopic culprit behind the debilitating fatigue, brain fog, and other persistent symptoms of long COVID: abnormal, sticky microclots embedded with neutrophil extracellular traps (NETs) in patients' blood. These microclots—tiny aggregates of clotting proteins—are small enough to obstruct the body's tiniest blood vessels (capillaries), restricting oxygen delivery to tissues and organs without triggering obvious large-scale clotting events. In long COVID patients, researchers observed a dramatic ~20-fold increase (median 19.7 times higher) in the number of these microclots compared to healthy controls, with the clots also tending to be larger. What sets this finding apart is the discovery that these microclots are structurally intertwined with NETs—web-like structures of DNA, enzymes (such as myeloperoxidase and neutrophil elastase), and proteins released by neutrophils (a type of white blood cell) to ensnare pathogens. Normally, NETs form temporarily and then dissolve, but in long COVID, they persist and become physically embedded within the microclots, creating highly resistant, "gummy" structures that evade the body's natural clot-breaking processes (fibrinolysis). This creates a chronic thromboinflammatory state, where blocked microcirculation and ongoing low-grade inflammation may sustain symptoms like exhaustion and cognitive impairment. The differences were so pronounced that machine learning models analyzing anonymized blood samples (via fluorescence microscopy for markers like ThT for amyloid-like structures, DNA stains, and MPO for NETs) could distinguish long COVID patients from healthy individuals with 91% accuracy—offering a potential objective biomarker for a condition that has long evaded reliable diagnosis through standard tests (e.g., normal D-dimer, PT/INR, or aPTT levels despite significant microclot burden). This work, led by teams including Prof. Etheresia Pretorius (Stellenbosch University) and Dr. Alain Thierry (Montpellier University), reframes long COVID as a tangible, blood-based disorder driven by dysregulated coagulation and innate immunity rather than vague "post-viral malaise." Targeting NETs or microclots—perhaps with therapies to degrade NETs or prevent their stabilization—could open doors to treating root causes instead of merely alleviating symptoms. [Thierry, A. R., Usher, T., Sanchez, C., Turner, S., Venter, C., Pastor, B., Waters, M., Thompson, A., Mirandola, A., Pisareva, E., Prevostel, C., Laubscher, G. J., Kell, D. B., & Pretorius, E. (2025). Circulating Microclots Are Structurally Associated With Neutrophil Extracellular Traps and Their Amounts Are Elevated in Long COVID Patients. Journal of Medical Virology, 97(10), e70613. DOI: 10.1002/jmv.70613]

Unless you speak up, soon you won’t be allowed to use a computer or phone without scanning your face and sharing your ID to some honeypot (which will eventually be hacked).

When simulation becomes the norm, it weakens the human capacity for discernment. As a result, our social bonds close in upon themselves, forming self-referential circuits that no longer expose us to reality. We thus come to live within bubbles, impermeable to one another. Feeling threatened by anyone who is different, we grow unaccustomed to encounter and dialogue. In this way, polarization, conflict, fear and violence spread. What is at stake is not merely the risk of error, but a transformation in our very relationship with truth.

Didn’t expect the Pope, of all people, to deliver the best explanation of radicalisation in the age of AI. But here we are. 👇

As we head towards midterm elections, NYT says it has identified “hundreds” of fake AI-generated pro-Trump social media accounts. Trump has reposted content from at least one of the fake accounts.

Maybe another explanation why people with #mecfs and #longcovid experience insomnia and other sleep disorders? Sleep pressure builds from mitochondrial electron leak in specific brain cells… ⬇️⬇️⬇️

People born during the 1918 Spanish Flu had a 2-3x increased risk of developing Parkinson’s compared to those born before 1888 or after 1924. COVID has infected billions. We will likely see a massive surge in neurodegenerative disease.

Y’ALL. This is not a drill. The Wayback Machine is often the only source of webpages that have been scrubbed from the Internet.

Reminder that Dr. Fauci said to have COVID under any kind of control we would need to have less than 10k infections daily. Our “low” now is 200-300k daily. Nearly on par with what we used to consider “high” and trigger enforced masking and distancing. It’s not over.

When my colleagues and I became severely sick and disabled over the past year, I knew I had to spring into action to learn about the mechanisms behind Long COVID. Our doctors were not giving us answers because the risks and drivers behind persistent spike are poorly understood and underecognized in medicine--the perfect recipe for disaster, disease, and mortality. I have been researching these mechanisms for the past year--perhaps my most impactful work in neurodevelopmental medicine was while on short-term disability leave while I try to determine how to stay alive and recover as a person with hypermobile Ehlers Danlos Syndrome (only diagnosed in December at age 36) and ADHD. Hopefully my next poster is a research-informed protocol targeting all of the multisystem factors indicated in section 4!

YA'LL I FOUND A SOURCE FOR MY POTS. WITH OVER 50% VEIN COMPRESSION, I AM ELIGIBLE FOR STENT PLACEMENT. This causes Sinus tachycardia Exercise intolerance Pelvic pain Back pain Pain with intercourse @Dr_Sudi was so sweet and let me cry because I could finally see what was causing my issues without seeming like a psychosomatic young woman who just needed therapy. I had passed my tilt table test and Raynaud's Test, and it was so disheartening and frustrating to me because I couldn't figure out what was wrong after consulting 15 doctors.

This is the bombshell that forced Melania into urgent damage control. Shit’s about to pop.

This is what happens when teachers are some of the most impacted by the 🦠 that shall not be named. FYI: Do not leave your health up to the same people who decided you are expendable. You will just get replaced.

Highly practical. Start today. Lymphatic and glymphatic techniques can be learnt and used even when bedridden. This is not woo woo crap. I know its hard to get just how powerful such simple interventions can be - but they really are that powerful. Dont ignore this!!

🔬Study shows SARS-CoV-2 causes direct damage to heart cell mitochondria - even months after recovery - helping potentially explain Long COVID heart symptoms like chest pain, palpitations & fatigue. Let’s break it down 🧵

One patient - a healthy 30-year-old man - collapsed during exercise due to cardiac arrest. He had no blocked arteries and no heart disease history. Biopsy revealed severe mitochondrial damage and mild myocarditis. He was 5 weeks post-COVID at the time.