MetenIsWeten
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Tel Aviv Mayor Ron Huldai: "We are destroyed. We are living in shelters for weeks now. Why exactly are we the ones suffering right now? We are the chosen people!" Life's tough, get a helmet.
Okay Jeff! that wasnt what I said, x.com/MeasslainteIRL… Okay, let’s be clear about what the Ontario study actually is and isn’t. It’s a large administrative database analysis death certificates, hospital records, ICD codes on about 6.4 million people aged 12‑50. It found lower odds of sudden death among the vaccinated (aOR 0.57) and no signal in the self‑controlled case series during the six weeks after a dose. Defenders call it “definitive proof” that mRNA vaccines don’t cause sudden cardiac death. But a perfectly executed study can still ask the wrong question. And this one is structurally blind to the very mechanisms that generated safety concerns in the first place. Documented pathways include persistent vaccine‑derived spike protein in cardiac tissue—found in autopsies and biopsies weeks to months post‑vaccination. Spike induces amyloid microclots that resist fibrinolysis and are invisible to standard D‑dimer tests. Microvascular ischemia can cause cardiac damage without elevating troponin or showing on angiography. Subclinical myocarditis can leave scar tissue that creates an arrhythmogenic substrate capable of triggering fatal events months later. Then there’s DNA contamination Speicher et al. documented 815‑fold variance between lots, with some batches 627‑fold above regulatory limits, and a direct correlation between higher contamination and more adverse events. This study has zero tissue data, zero biomarker data, zero imaging, zero autopsies with specialized staining. It relies on ICD‑coded “sudden death” from administrative records and nearly 90% of those cases were out‑of‑hospital deaths without diagnostic confirmation. No spike testing, no amyloid stains, no DNA lot tracking. Calling those “cases” and then concluding safety is assuming what you’re supposed to prove. The study’s own data undermines its interpretation. Flu vaccination was associated with a 28% lower odds of sudden cardiac death. Does the flu vaccine prevent cardiac death? No that’s healthy vaccinee bias. The same bias applies to the COVID‑19 findings, but instead of acknowledging it, the study treats the flu result as a “control” proving their methods work, while ignoring what it actually demonstrates. Survivorship bias masquerades as a dose‑response relationship. One dose showed an aOR of 0.88; two or more doses showed 0.53. The interpretation that more doses mean more protection ignores the simple fact that people who die after dose one never get dose two. Dead people cannot be counted in the two‑dose group. The self‑controlled case series method is useful for time‑invariant confounders, but it’s not immune to time‑varying or event‑dependent bias. People get vaccinated when they feel healthy. Background risk changed across pandemic waves. And fatal events are inherently event‑dependent—the dead are by definition unvaccinated near the time of death. As Clare Craig put it, SCCS cannot address the selection bias where dead people cannot be vaccinated. Removing the deceased from the analysis artificially reduces apparent risk. Then there’s the timeframe problem. The study limits its analysis to six weeks post‑vaccination, but documented spike persistence in cerebral arteries reaches 17 months, spike in circulation up to 709 days, amyloid microclot progression spans months to years, and DNA contamination effects if they matter would show on a timeline of 5‑10 years. Claiming “no long‑term risk” from a six‑week analysis is not science; it’s ignoring documented timelines. The study treats vaccination as a binary yes/no, but batch‑level variance means grouping all lots together obscures the true risk. When one lot has 815‑fold more DNA contamination than another, and higher contamination correlates with more adverse events, averaging across lots hides the signal. Standard clinical tests are blind to amyloid microclots. Spike binds fibrinogen and induces an amyloid transformation that resists fibrinolysis, so D‑dimer comes back normal. Patients present with symptoms, get a normal D‑dimer, are told there’s no clotting issue, their symptoms are dismissed, and months later they die. The death certificate says “unknown cause” or “cardiac arrest,” and the Ontario study counts that as a negative data point. That’s circular reasoning: using diagnostic codes from a system that systematically misses the mechanism to claim the mechanism doesn’t exist. So where does that leave us? The study can say there was no massive, obvious population‑wide spike in coded sudden deaths detectable by this method. That’s a useful data point. But it cannot refute mechanistic concerns, rule out smaller or rarer or delayed effects in subgroups, or prove cardiac safety against spike‑related pathways. In the face of documented biological mechanisms spike persistence, amyloid microclots, batch contamination, dose‑response with adverse events the burden of proof shifts. Demonstrating safety requires tests that can actually engage with those mechanisms: tissue testing, amyloid‑specific stains, lot‑tracked outcomes, long‑term follow‑up with proper autopsies. Treating one administrative database study as the final word while waving away the biology is not rigorous science. It’s appeal to authority dressed up as “the study was perfectly designed.”
🏮 Another population-level study finds no evidence whatsoever of that healthy younger individuals given COVID-19 vaccines had increased risk of "dying suddenly", i.e. with sudden cardiac deaths. Researchers in Toronto just published a study in PLOS Medicine assessing whether risk of sudden cardiac death was increased after COVID-19 vaccination in healthy young people 12-50yrs old. The study was a population-based matched case-control study from a cohort of 14,966,193 residents of Ontario, Canada, focusing on the subset of 6,365,451 who were <50yrs and "healthy", with no medical history of cardiovascular disease, cancer, diabetes, mental illness, dementia, COPD, IBD, chronic liver or kidney disease, autoimmune disease, or alcohol or illicit drug use. Cases included 4,803 individuals from that cohort who "died suddenly" between April 1, 2021 and June 30, 2023, meaning they either died outside the hospital or died in ER or within 24hr of admission with indication of cardiac arrest, sudden death or arrhythmia but no indication of trauma, mental illness or substance use. Controls included 24,030 individuals matched 5:1 to cases in terms of age, sex, geographic area, and neighborhood income quintile, with their index data set to death date of matched case. After matching, all of these variables were well-balanced between cases and controls. The primary analysis was a conditional logistic regression to assess association of case/control status with vaccination status (any vaccine), with covariate-adjustment for confounders including COVID-19 infection, influenza vaccination, number of SARS-CoV-2 PCR tests to estimate COVID healthcare utilization, and history of asthma, hypertension and mood/anxiety disorders. Many sensitivity analyses were done including separate analyses for vaccine types, focusing on the 6wk period after vaccination, focusing only on ER/hospital deaths with cause well-documented, excluding any opioid deaths, focusing on <=40yrs old, plus a secondary self-controlled case series comparing the 6wk after doses 1/2/3 to other time periods to perform an assessment free from any subject-level residual confounders such as healthcare utilization or other factors related to healthy vaccinee effect. In the primary analysis, 77.1% of controls received any COVID-19 vaccine before the index date, while only 67.4% of cases did, which resulted in an adjusted odds ratio of 0.57 (95% CI 0.53-0.61), suggesting a 43% reduced risk of sudden death in the vaccinated group. All secondary analyses found similar results, with vaccinated having lower risk of sudden death than unvaccinated, with some statistically significant and some not. None of the analyses had any indication whatsoever that risk of sudden death was increased after vaccination, either in the short or long term. This validates results from two earlier UK studies using self-controlled case series that found no increased risk of sudden cardiac death within 4wks (Nafilyan et al. 2023 Nature Communications PMID: 36973247) or 12wks (Xu et al. 2024 Vaccine PMID: 38388239) after COVID-19 vaccination. So, in spite of all of the repeated claims on social media and in videos purporting major increased risk in sudden cardiac death after COVID-19 vaccination, the population-level data continue to strongly refute these claims.
Omdat wij weten wat bezetting en onvrijheid betekenen, begrijpen we wat Oekraïne doormaakt. Rusland gebruikt grof geweld. Dit is geen ver-van-ons-bed-show – het raakt direct onze veiligheid. Daarom moeten we Oekraïne blijven steunen.
Flikker op met je Oekraïne. Tijd voor de noden van de eigen bevolking!
RFK Jr. reveals why he stopped getting the flu shot: “I was getting a flu shot every year.” “I stopped in 2005 when I began looking at the side effects.” “One of the injuries that was listed on a lot of them was spasmodic dysphonia, which is an injury I have to my voice.” “That turns out to be a vaccine injury.” “That’s why my voice is so screwed up.” “Do I know that it was caused by my annual flu shot? I have no idea.” “It’s a potential culprit that I cannot rule out.” “We should have that data, but we don't.”
‼️MUST LISTEN‼️ Dr. Peter McCullough joins Theo Vonn to discuss the heartbreaking truth that nobody is talking about.... The COVID 💉 is directly linked to myocarditis. Period.
"De Amerikaanse minister van Volksgezondheid Robert F. Kennedy Jr. vertelt waarom hij gestopt is met het griepvaccin: “Ik liet me elk jaar tegen griep vaccineren.” “Ik ben er in 2005 mee gestopt toen ik me ging verdiepen in de bijwerkingen.” “Een van de bijwerkingen die bij veel vaccins werd genoemd, was spasmodische dysfonie, een aandoening die ik aan mijn stem heb.” “Dat blijkt een vaccinatiegerelateerde aandoening te zijn.” “Daarom is mijn stem zo verpest.” “Weet ik zeker dat het door mijn jaarlijkse griepprik kwam? Ik heb geen idee.” “Het is een mogelijke oorzaak die ik niet kan uitsluiten.” “We zouden die gegevens allang moeten hebben, maar die hebben we niet.”"
Myocarditis is Caused by COVID-19 Vaccination not SARS-CoV-2 Infection Alone and It's Fatal M. Nathaniel Mead, Jessica Rose, William Makis, Kirk Milhoan, Nicolas Hulscher and Peter A. McCullough. Myocarditis after SARS-CoV-2 infection and COVID-19 vaccination: Epidemiology, outcomes, and new perspectives. INTERNATIONAL JOURNAL OF CARDIOVASCULAR RESEARCH & INNOVATION. Jan-Mar 2025, VOL. 3, ISSUE 1, pp. 1-43, DOI 10.61577 ijcri.2025.100001 reseaprojournals.com/journals/cardi…
