Slobodan Ponjevic

First Kucoin, Now Binance Eventually the world will follow You are $NAT ready

NEW ARTICLE: IVERMECTIN, FENBENDAZOLE, HBOT Testimonial - 70 year old NEW ZEALAND man with Stage 4 Pancreatic Cancer given 2-3 months to live, reports after 1 year - NED! No chemo, radiation or Oncology. Ivermectin Cancer Revolution is catching on in beautiful New Zealand! 🇳🇿😃 STORY: 70 year old NEW ZEALAND man with Stage 4 Pancreatic Cancer given 2-3 months to live In May 2025 he started Ivermectin, Fenbendazole, HBOT. No Oncology treatment Results after 12 months: No Evidence of Disease (NED) There are now dozens of testimonials of Stage 4 Cancer patients becoming NED without any conventional Oncology treatment. These are the cases where the repurposed drug skeptics simply go silent, because they have no explanation to offer. I always encourage Cancer patients to work with their Oncologist and consider all mainstream Cancer Treatments. Always. But if we believe in bodily autonomy, a concept most doctors today don't believe in, there is not only a "Right to Try", but also a "Right to Choose". Cancer patients have the right to make decisions about their treatments and their own bodies. I have helped over 9000+ Cancer patients with repurposed drugs: Ivermectin, Mebendazole and Fenbendazole in the largest Ivermectin Project in the world. We are bringing this Project to Florida! 😃 Please be patient during our move, as all consultations are currently halted. Our waitlist is in the thousands and growing daily. No one else is posting repurposed drug success stories because they don't have any. Many companies are trying to get on the Ivermectin and Mebendazole train after 2 years of my hard work, but they don't actually see or help cancer patients. Stay away from those who are selling overpriced Ivermectin and Mebendazole kits!

@binance NATing happens because you have NAT to support miners and it is already adopted with major mining pools. East is buying, west is sleeping to fomo in too late.

What happens when the last BTC is mined? A closer look at Bitcoin after the final coin is mined. Read more 👇 binance.com/en/academy/art…

NEW ARTICLE: RESEARCH: NICLOSAMIDE in CANCER and other diseases - 2025 Review Paper from Henan, China NICLOSAMIDE was approved by FDA in 1982 for treating tapeworm infections, and was designated an essential drug by WHO. NICLOSAMIDE in CANCER Mechanisms: 1. Mitochondrial uncoupling - this is its major anti-cancer mechanism Niclosamide acts like a mild “leak” in the mitochondrial membrane, letting protons slip across without making ATP. This increases oxygen use, lowers ATP levels, raises AMP/ATP ratio & activates AMPK (energy sensor) to inhibit mTOR (slows down cancer cell growth). Cancer cells, which often rely on inefficient energy pathways (Warburg effect), are especially vulnerable. This can lead to inhibition of ATP synthesis, exhausting cellular energy reserves, metabolic stress, and cell death. 2. Signal pathway modulation - It inhibits several “pro-cancer” pathways, including: - Wnt/β-catenin (important in cancer proliferation, especially colorectal). - mTOR (controls cell proliferation, growth and metabolism). - Notch pathway (controls proliferation, growth, angiogenesis and cellular migration) - STAT3, NF-κB, etc. (cancer survival, proliferation). - Promotes apoptosis (programmed cell death) and reduce inflammation. 3. Combination with existing chemotherapies 4. Addresses Multi-Drug Resistance - by enhancing production of reactive oxygen species. OTHER DISEASES - beyond cancer, Niclosamide’s versatility includes: Anti-bacterial - strong in vitro activity against resistant gram-positive bacteria. its antibacterial activity involves altering bacterial morphology, reducing intracellular ATP, and inhibiting α-HL secretion (Staph Aureus) it inhibits all three toxins of Clostridium difficile by targeting a host process essential for toxin entry into colonocytes. In a mouse model, it reduced both primary disease and recurrence without affecting gut microbiota diversity it demonstrates marked inhibition of vancomycin-resistant Enterococcus faecium (VRE) It effectively eliminates H. pylori adhesion/invasion Anti-Fungal - demonstrates impressive antifungal effects in vitro and in vivo niclosamide effectively disrupted and removed biofilms of drug-resistant Candida albicans and Candida auris by targeting mitochondrial respiration Anti-Viral - Niclosamide could have action against SARS-CoV, MERS, Zika, Hepatitis C, Ebola, EBV Niclosamide inhibits viral replication by reducing mTOR activation and prevents viral entry inhibits virion production in EBV-infected cells while showing cytotoxicity and inducing cell cycle arrest demonstrates strong activity against RSV in vitro, with a 94 % inhibition rate suppresses Hepatitis C replication and reduces viral load Metabolic disorders (e.g., Type II Diabetes, fatty liver): Mitochondrial effects and AMPK activation can improve energy balance and reduce fat accumulation. Diabetes: modulates metabolic pathways, improves insulin sensitivity Fatty liver (NASH, NAFLD) - reduces hepatic inflammation Neuropathic Pain - reduces inflammation by inhibiting NF-kB, STAT3 alleviates pain and nerve damage Rheumatoid Arthritis - inhibits NF-kB activation reduces secretion of IL-6 and other inflammatory cytokines, blocks migration and invasion of RA fibroblast-like synoviocytes. Psoriasis - inhibits keratinocyte hyperproliferation and reduces inflammation by downregulation of STAT3, NF-kB. Airway Diseases (Asthma, Cystic Fibrosis, COPD) Niclosamide inhibits certain Ca-activated Cl channels involved in mucus hypersecretion & bronchoconstriction (reduces mucus production & secretion. it blocks release of mucus and inflammatory mediators which reduces bronchoconstriction. anti-fibrotic agent for lung fibrosis - Niclosamide downregulates expression of collagen proteins and inhibits pro-fibrotic cytokines. Endometriosis - Niclosamide targets STAT3 and NF-kB, reduces proinflammatory cytokines and reduces endometriotic lesion growth. ALS In vivo experimentation involving two ALS mouse models demonstrated slowed disease progression, enhanced survival rates, and reduced motor neuron loss and muscle atrophy. At the molecular level, niclosamide inhibited dysregulated pathways such as STAT3 and mTOR, reduced gliosis, and dampened inflammatory signaling niclosamide has shown remarkable potential by efficiently inhibiting the expression of S100A4, a Ca2+-binding protein frequently upregulated in reactive microglia and astrocytes in ALS models. This inhibition contributes to reduced microglial reactivity, cytoskeletal changes & inflammation in ALS Systemic Sclerosis - inhibits STAT3, AKT, and Wnt/β-catenin pathways reduced immune cell activation reversal of skin and lung fibrosis and reduced rate of inflammation CONCLUSION: Ivermectin, Mebendazole and Fenbendazole are leading the repurposed drug revolution but Niclosamide is the most promising of the other anti-parasitics!

DeSantis is the only Governor that fought for the people during COVID, who has fought for medical freedom since then.

NEW ARTICLE: RESEARCH: MEBENDAZOLE in Cancer - 2025 Iran Paper - How Mebendazole overcomes Cancer Drug Resistance! 2025 Aliabadi et al - Critical dysregulated signaling pathways in drug resistance: highlighting the repositioning of mebendazole for cancer therapy Cancer drug resistance significantly reduces the effectiveness of current anticancer treatments This highlights the need for developing new multi-targeting drugs that are more cost-effective, have fewer side effects, and remain effective against cancer. Drug repurposing offers a promising solution over expensive targeted therapies. Repurposing Mebendazole: it’s already been given to millions of people at high doses with a very good safety record, repurposing it for cancer is much faster and cheaper than inventing a brand-new drug. How MEBENDAZOLE Fights Cancer and Cancer Drug Resistance. This review breaks down six key ways Mebendazole attacks cancer. Mebendazole hits all of them at the same time, which is why it may overcome resistance better than single-target drugs: 1. Disrupts the cancer cell’s skeleton (microtubules) MBZ binds to the same spot on tubulin that the cancer drug colchicine uses. This stops cancer cells from properly dividing (they get stuck in the G2/M phase of the cell cycle and eventually die).It’s similar to some existing chemotherapies but appears safer. Starves the tumor of blood supply (anti-angiogenesis) 2. Tumors need new blood vessels to grow and get nutrients. MBZ blocks a key signal called VEGFR2, so the tumor can’t build those vessels. 3. Triggers programmed cell death (apoptosis) MBZ tips the balance of proteins inside the cell (raising “death-promoting” ones like Bax and p53, lowering “survival” ones like Bcl-2 and XIAP). This activates enzymes called caspases that make cancer cells commit suicide in an orderly way. 4. Forces cells to “eat” themselves (autophagy) Autophagy is the cell’s recycling system. MBZ can ramp it up so cancer cells digest their own damaged parts until they die or become easier to kill. 5. Mebendazole wakes up the immune system It shifts immune cells called macrophages from a “tumor-friendly” state (M2) to a “tumor-killing” state (M1 type). It also increases signals that attract other immune attackers and works especially well when combined with radiation. 6. Stops cancer from spreading (anti-metastasis and MMP inhibition) Cancer cells use enzymes called matrix metalloproteinases (MMPs) to chew through surrounding tissue and travel around the body. MBZ blocks them. Additionally, MBZ calms down several overactive growth pathways that cancers use to survive and resist drugs (examples: Hedgehog, ERK/MEK, MYC, NF-κB, AKT, etc.). What the evidence shows Lab dishes (in vitro): Very low concentrations (often under 1–5 micromolar) kill or slow down breast, colon, lung, stomach, ovarian, melanoma, and brain cancer cells. MBZ works even better when paired with standard chemo or radiation. Mice (in vivo): Oral doses (10–50 mg/kg) shrink tumors, reduce the number of polyps in colon cancer models by up to 90 %, slow metastasis, and improve survival. Tumors weighed far less (e.g., one study: ~2 g vs. 12 g in untreated mice). Humans (early clinical trials): Small studies in brain tumors (Glioblastoma) and gastrointestinal cancers showed it is safe even at high doses (up to 4 g/day). One glioma trial reported median survival around 21 months when combined with standard treatment. The main practical challenge (and the fix) MBZ doesn’t dissolve well in water and isn’t absorbed very efficiently from the gut into the bloodstream or tumors. The review discusses new “drug delivery” solutions that researchers are testing: polymeric nanoparticles nanostructured lipid carriers micelles nanosuspensions These make Mebendazole more soluble, help it reach the tumor, and may let doctors use even lower doses. Main Takeaways from the Paper: Mebendazole is a strong candidate for drug repurposing in cancer because it: 1. attacks cancer from many angles at once (great for beating resistance), 2. is already proven safe and cheap, 3. works in many different cancer types in the lab and in animals, 4. shows early promise in people, 5. and can be combined with existing treatments. CONCLUSION: Overall, the paper is optimistic: Mebendazole could become a low-cost, low-toxicity addition to the cancer-fighting toolbox, helping patients whose tumors have stopped responding to standard therapies. In short, this review makes a detailed, evidence-based case that an old “worm pill” might help solve one of the toughest problems in modern cancer treatment - drug resistance - by hitting the cancer where it hurts in multiple ways at the same time.

🇪🇺 The EU Plans to Crack Down on VPNs: "The new EU age ID verification system will not be 'bypassed' via VPN access." They couldn’t stop illegal migration, but suddenly becomes North Korea when it comes to controlling what Europeans read online...

@thepiratebaybtc @Rogerwilco66255 @natgmi Yes but this is public info, it is like mining, so yes you are right it is not classic mining but this changes nothing because NAT is completely linked to BTC mining and there is no NAT without mining BTC. That is the reason NAT exploded and miners adopt.

@Rogerwilco66255 @natgmi Stop support posting misinformation about $NAT ZERO HASH

⬛️ 65% hashrate mining $NAT. ⬛️ F2Pool & SpiderPool distributing. ⬛️ CEXs loaded. ​Next: 🔜 100% grid adoption. 🔜 Binance & Coinbase forced to market buy the base layer. ​NATstoppable. 🦇

🇺🇸 Senator Ron Johnson just dropped a damning new report. Biden health officials knew their own VAERS system was hiding serious safety signals from COVID vaccine injuries. They were shown a much better algorithm that actually flagged dangerous adverse events, but they refused to use it and stuck with the old one. Here are the 5 biggest things it reveals: An FDA scientist created a much better system that actually caught serious side effects the official FDA method was hiding. She showed her findings to top officials starting in March 2021, but they refused to use the better system and told her to stop her work. The improved system flagged real danger signals for sudden heart attacks, blood clots, sudden death, Bell’s palsy, and other serious problems. Instead of warning the public, FDA leaders were more worried about “anti-vaccine rhetoric” than protecting Americans. Even after seeing clear warnings, the FDA failed to promptly alert doctors and the public about the risks. Source: @SenRonJohnson

It feels like the world doesn't care about the Lebanese people. Villages are being leveled, yet the world is silent

@tap_protocol @Greekdx Thanks, it worked. Originally I didn't want to touch that options so that I don't lose NAT in with some error, but it seems that NAT is safe with this or we should be careful about some part of this process?

@PonjevicS @Greekdx Here is a video tutorial on how to use the BTC reclaim feature.

Best wallet for Ordinals? I used to use Leather but they are sunsetting Ordinals...

They are doing it. The EU now wants to accelerate the rollout of the age verification app so that all member states make it available by the end of this year. It can be used as a it's own app or be integrated into digital ID. Will you be using it?

@tap_protocol @Greekdx I very much appreciate your quick reply but I stil don't know how to unlock btc on tap wallet. With NAT I managed but with BTC I don't see any option to do that.

We've just tested this on both mobile and browser, and the sats reclaim feature is working well as usual. Once you've marked some inscriptions as 'spendable', wait 10-20secs, refresh the app (mobile or browser) and you will see the new available BTC added to your balance when you click on the 'Send' button and enter that menu (top right hand corner).

@tap_protocol @Greekdx Please make BTC unlock usable option in tap wallet. Unusable/locked btc is very irritating and will result many leave TAP wallet.

@Greekdx We invite you to try TAP Wallet x.com/tap_protocol/s…

🔥 This is an important moment. F2Pool didn’t just integrate the protocol, they showed the entire industry what the future of mining looks like. $BTC + $NAT in one block. Two native rewards. No forks. No protocol changes. This is only the beginning .

This is big! Nice idea is now reality! Buy $NAT early, not when everyone talks about it in a few years.

🚨 $NAT is NOT just another token miners earn. It is the first and only native Bitcoin second subsidy in history: • Automatically derived from the “bits” field of every BTC block (non-arbitrary via DMT) • Rewarded DIRECTLY to the miner who finds the block (same hash, same block, same address) • Zero extra effort True infrastructure asset strengthening #Bitcoin security forever. $DMT $NAT #BitcoinMining

Just like COVID, we need this fake crisis so that more people are forced to wake up to the reality that the system is pure theatre

🚨🇪🇺 EU WANTS YOUR SEARCH HISTORY - EYE OF SAURON Every search, click, scroll… ALL OF IT They say “anonymous”. NOTHING IS Your health, beliefs, money problems, PRIVATE life… exposed Data sent to THIRD PARTIES One leak and it’s OVER