Ben Noor, MD

141 posts

Ben Noor, MD

Ben Noor, MD

@bennoormd

Podcast Enthusiast. Current cardiology fellow at Harbor-UCLA. Tweets are my own

Katılım Haziran 2020
227 Takip Edilen160 Takipçiler
Ben Noor, MD
Ben Noor, MD@bennoormd·
@drjohnm Often, new AF coincides with a lot of non-cardiac acute issues in the hospital. It preserves your ability to cardiovert if needed without TEE while also giving flexibility to stop AC if needed for clinical instability or procedures
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John Mandrola, MD
John Mandrola, MD@drjohnm·
Why do people use heparin or enoxaparin for acute AF? It drives me bananas. There’s zero data!
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Ben Noor, MD
Ben Noor, MD@bennoormd·
@Bgheyath Why confirm a wedge sat if you have a clear PAWP waveform?
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Bashaer Gheyath MD FACC 🫀👩🏻‍⚕️
🫀 PCWP: Technique Matters Step 5: PCWP— Advance distally with balloon up, then deflate ~50% while advancing. ⚠️ Dampened waveform? • Disconnect • Aspirate • Flush • Re-check Persistent damping → consider larger-lumen catheter. Always confirm with >95% saturation.
Bashaer Gheyath MD FACC 🫀👩🏻‍⚕️ tweet mediaBashaer Gheyath MD FACC 🫀👩🏻‍⚕️ tweet media
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Bashaer Gheyath MD FACC 🫀👩🏻‍⚕️
🧵 Golden Thread: Dr Nishimura’s Stepwise Approach to Right Heart Catheterization - IVC / RA Entry Step 1: Start in the IVC → RA Advance & turn away from the hepatic veins. Obtain a venous sat early to exclude a major left-to-right shunt. #ICSC2026
Bashaer Gheyath MD FACC 🫀👩🏻‍⚕️ tweet mediaBashaer Gheyath MD FACC 🫀👩🏻‍⚕️ tweet media
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Kayiira Mubaraka
Kayiira Mubaraka@MubkayiiraMD·
@NephroP 1. Parasternal short axis view - aortic level 2. Thrombus most likely 3. Chamber is RVOT( Right ventricular outflow tract).
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NephroPOCUS
NephroPOCUS@NephroP·
⭕️ What view? ⭕️ (likely) abnormality? ⭕️ Which chamber is the abnormality located in? #POCUS #FOAMed #FOAMcc Source 🔗 CASE (Phila). 2019;3(4):167-170. doi: 10.1016/j.case.2019.04.001.
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Abdulla A. Damluji, MD, PhD
Reevaluating Beta-Blocker Recommendations Post-Myocardial Infarction: Perspective on the 2025 Guideline Update: @JACCJournals 🥸Commentary on the guidelines - early beta blocker use post MI - 😱@MichaelGNanna wrote extensively on this in older pts. 🥸Take a look 👇👇👇
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Kiran Rikhraj
Kiran Rikhraj@KiranRikhraj·
#POCUS TEE pearl: Keep it simple. While learning TEE, I felt overwhelmed by the number of views and angles. But from a POCUS lens, you only need a few views to get enough data to manage your patients. I'm not an expert by any means and wanted to share the 4 views I focus on:
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Ahmed Ata
Ahmed Ata@Ahmedata7777·
AKI: pre renal vs renal (ATN) Taken from Passmedicine What you most use to differentiate in clinical practice?
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Dr. Filippo Cademartiri
Dr. Filippo Cademartiri@FCademartiri·
PHOTON COUNTING CT A taste of the comparison between PCCT and conventional CT (EID) in the same patient Photon Counting CT determines a whole new sense through which we can look at infinitely small features of the human body and their alterations that lead to diseases. It is finally like having a microscope that works on every part of the human body delivering new information, more accurate and reliable information, and because of this, more precise and individualized medical care for each patient. The immediate result is in that using it as a primary tool means no/less need for any further testing, optimal characterization of pathologic alterations, the possibility to monitor subtle changes in disease precursors of disease during preventing therapies, and so forth. A new era is coming into practice and it is the age of Photon Counting CT which pushes this boundaries further away. PCCT is a NEW Imaging Modality. PCCT is changing the game, the field, the language, the priorities and in the end it will change the entire infrastructure of diagnostic medicine. #CardiacImaging #MedicalInnovation #StentAssessment #Radiology #PCCT #photoncounting #QuantumHD #CT #computedtomography #yesCCT #coronaryarterydisease #ischemia #naeotomalpha #Peak #Pro #Prime #speed #cardiac #highresolution #siemenshealthinners #CardiacCT #PhotonCountingCT #MedicalImaging #HeartHealth #CardiovascularInnovation #Radiology #AIInMedicine
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Michelle Kittleson MD PhD
Michelle Kittleson MD PhD@MKIttlesonMD·
76F has lipid panel: cholesterol 254, HDL 89, LDL 145, triglycerides 92. ASCVD 10-year risk 21.5%. Also needs MV repair for MVP with severe MR so CTCA ordered: no evidence of coronary artery disease and coronary artery calcium score zero. What do you recommend?
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Eric H Yang, MD
Eric H Yang, MD@datsunian·
No greater privilege to come back to the place of your upbringing & why you do what you do 🙏 @HarborUCLA Cardiology for giving me the chance to chat all things imaging & #cardioonc for GR Great to see my teachers, mentors, cofellows, friends, & the future of ❤️ #harborstrong
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Alfonso Valle
Alfonso Valle@ValleAlfonso·
⚠️Angiotensin Receptor Neprilysin Inhibition Across Ejection Fraction and Acuity Spectra in #HF via @pmyhre & @lamcardio 👉PARADIGM-HF, 👉PARAGON-HF 👉PIONEER-HF 👉PARAGLIDE-HF, 📌 covering all 4 corners of the stage C HF acuity-by-LVEF spectra 📌clear benefit of ARNI over ACEI/ARB in both the subacute and chronic settings up to an LVEF<60% @JACCJournals 📁 jacc.org/doi/10.1016/j.…
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Ben Noor, MD
Ben Noor, MD@bennoormd·
@AnilMakam @JeremySussman I think “goal-directed” is a common misconception. It is “guideline-directed,” but many people mix it up with “goal-directed,” which is in reference to septic shock resuscitation in the era of the Rivers trial.
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Anil Makam
Anil Makam@AnilMakam·
@JeremySussman now that you say it, I'm sure goal directed is also widely inferred
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Anil Makam
Anil Makam@AnilMakam·
Love these polls to see practice variation. Just wow. I dislike the term GDMT since implies this is scripture for all patients at all times I would do: DAPT risk factor modification, including SGLT2i or GLP1ra if indicated but no further 'GDMT' intensification since not 'GD'
Michelle Kittleson MD PhD@MKIttlesonMD

53M with EF 40% and LAD disease. Undergoes LAD PCI and initiation of ACEI and BB. Follow-up echo 3m later with EF 50%. NYHA Class I, BP 110/70, normal exam, K, Cr, BNP normal. Now referred to you for GDMT optimization. What do you do?

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Ben Noor, MD
Ben Noor, MD@bennoormd·
@nyulaw As an NYU alumni, I’m ashamed of the SBA president’s message. My hope is that the rest of the law school students and staff come out and openly condemn her statements supporting terrorism.
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Ben Noor, MD retweetledi
Ryan B. Cohen
Ryan B. Cohen@ryancohenmd·
Happy to present our work on de novo pulmonary embolism formation at #ACS2022
Ryan B. Cohen tweet mediaRyan B. Cohen tweet media
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Ben Noor, MD
Ben Noor, MD@bennoormd·
@NealDixit @VivekKulkarniMD Great points. I think it's ultimately not straight forward, so I'd lean towards less invasive route. But looking at CAC score, which is more predictive of future events than # of stenoses, comorbidities, surgical risk, status of prior RCA stent may push me towards surgery
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Neal Dixit, MD
Neal Dixit, MD@NealDixit·
@bennoormd @VivekKulkarniMD See FAME 3 93% on statin. No SGLT2i or PCSK9i but maybe benefits additive to CABG. In CABG trials often angina OR ischemia. If most MI is caused by non-flow limiting stenosis and angina caused by flow limiting stenosis then symptoms =/= MI/death risk. CABG bypasses disease ⬇️risk
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Vivek Kulkarni MD
Vivek Kulkarni MD@VivekKulkarniMD·
~50M w/ remote RCA NSTEMI s/p PCI, LVEF 45-50%, and DM2. No symptoms. Prior doc ordered routine post-PCI treadmill SPECT: ~12 min Bruce, no symptoms, but large area of mild inf ischemia. If CCTA/ICA shows multi-vessel CAD, but he has no symptoms at all, should he get CABG?
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Ben Noor, MD
Ben Noor, MD@bennoormd·
@NealDixit @VivekKulkarniMD I always find this topic difficult. Benefit of CABG in this population is based on old data without contemporary meds. Also, patients were included if they had angina or ACS at time of CABG. So hard to extrapolate benefit to this patient.
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