LLicit
5.3K posts


Pasteurized milk is a processed food
I used to think raw milk was simply about being more nutritious, which is certainly true
I have come to realize that the enzymes are even more important
Lactase, lactoferrin, amylase, catalase, ect… all destroyed by pasteurization
These enzymes are essential in metabolizing the milk and important for overall health
Here are some of the enzymes in milk and there properties:
- Catalase: Acts as an antioxidant, breaking down hydrogen peroxide into water and oxygen to prevent oxidative stress within the milk
- Amylase: Assists in the breakdown of starch into sugars
- Lactoferrin: immune system boosting, designed to protect the nursing young from infection, protects you from getting sick from any contaminants in the milk
- Lactoperoxidase: one of the most abundant enzymes, anti-microbial, inhibits the growth of a wide range of bacteria, including Salmonella and Staphylococcus aureus
- Lipase: essential for lipid metabolism, responsible for the hydrolysis of milk fats into free fatty acids and glycerol
These enzymes prove that milk should be raw, the way God intended
Lactase is the same enzyme they give lactose intolerant people, found in REAL milk
Other enzymes literally prevent any kind of pestilence the milk could have
Milk is perfect the way it is

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@SamaHoole Many diets can reverse diabetes via weight loss
Something like Mediterranean a better approach as then you are not just swapping diabetes for heart disease
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Your doctor will not tell you that Type 2 diabetes is, in many cases, fully reversible.
Not manageable.
Not slowed down.
Reversed.
Virta Health published peer-reviewed data showing that a low-carbohydrate dietary intervention reversed Type 2 diabetes markers in 60% of patients within one year, with 94% reducing or eliminating insulin.
This costs nothing.
The global diabetes drug market was worth $67 billion in 2023.
Your doctor is not incentivised to discuss dietary reversal.
Your doctor is not trained in dietary reversal.
Your doctor's practice receives QOF payments for managing your diabetes.
Management means medication.
Medication is a recurring revenue model.
Reversal is a one-time event.
This is the healthcare system working exactly as designed.
It was not designed to reverse anything.
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@animaldocfilm Can you post supporting information for this
(as runs counter to the usual narrative)
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@AlpacaAurelius Unless the study accounted for:
1. Survivorship bias
2. Reverse causality
3. Effect modification
Then highly misleading ...
Better to focus on the much more powerful interventional studies (which consistently show LDL ⬇️ = CVD ⬇️)
europepmc.org/article/med/36…
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@SamaHoole Odd how influencers always post easily confounded observational studies (like this one - survivorship bias, reverse causality & more)
They never-ever mention the much more powerful interventional studies (which consistently show LDL ⬇️ > CVD ⬇️)
Doesn't suit the sell I guess
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In populations over 70, higher LDL is associated with lower all-cause mortality in multiple large studies.
Not higher. Lower.
The relationship inverts precisely at the age when the hypothesis predicts it should be most dangerous.
The study of 68,000 elderly patients published in the BMJ Open in 2016 found that in 80% of the cohorts examined, total cholesterol and LDL had an inverse or no relationship with mortality.
The higher the LDL, in older populations, the longer they tended to live.
This is the wrong finding for the hypothesis.
It gets cited infrequently.
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@SamaHoole It's been known for ever and a day that heart disease is multifactorial
LDL-C is an important risk factor but no one ever said it was the only risk factor

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PCSK9 inhibitors can get LDL to single digits.
Ten. Twelve. Fifteen mg/dL.
Levels that make a standard "high" LDL of 180 look enormous.
If LDL is the cause of heart disease, this dose-response relationship should produce near-elimination of cardiovascular events.
It doesn't.
The residual risk remains substantial.
You've removed the supposed cause.
The disease is still there.
There are two possible responses to this.
One: maybe LDL isn't the primary cause.
Two: lower it even more.
The pharmaceutical industry has chosen the second response.
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@PaulReynoldsPhD Advanced glycation end products (AGE's) in food
precisionnutrition.com/all-about-cook…

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@LogicRoad @bigfatsurprise It might be useful to watch this
Three ways studies can be designed to make it appear saturated fat is harmless
youtube.com/watch?v=mBFe1Q…

YouTube
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Even if this were true, it isn't, there are 100's of other books and scientific studies, metanalysis that all show overwhelming data showing there is no benefit to removing or reducing animal fat and protein from the diet.
This is key here because vegans like to focus on one single thing that shows a weak association and ignore the millions of other points of data, information and common sense.
This reminds me of how a young earth creationist will use a single piece of information, like a tree found through multiple layers of earth that should not be possible, as 100% proof all while ignoring the millions of other fossils, layers etc. showing the earth is very old.
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Confused about saturated fat? This book published a decade ago detailed the politics, science and history of this controversial fat.
Ralph M. Prescott@rmprescott
@JoshuaLelon @grok @seconds_0 @goblinodds @bigfatsurprise has written a lot on the reasons why saturated fats (used since prehistory) became vilified in the late 20th century.
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@LogicRoad @bigfatsurprise That's incorrect. Dietary cholesterol does impact plasma levels as below
For most people the increase is relatively modest but an increase nonetheless (which is important for #1 killer)
As per other post, it can be substantial for hyper-absorbers (approx. 1 in 5 people)

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Even the godfather of the diet heart hypothesis, which is what 100% of the anti saturated fat and cholesterol misinformation stems from admitted that there is no connection between cholesterol in the food and cholesterol in the blood.
“There’s no connection whatsoever between cholesterol in food and cholesterol in the blood. None. And we’ve known that all along. Cholesterol in the diet doesn’t matter at all unless you happen to be a chicken or a rabbit.” - Ancel Keys
FYI the study that helped start the cholesterol misinformation was done on rabbits, the same study was replicated on dog which have a much more similar digestion system compared to humans and guess what, there was no negative affects of cholesterol for the dogs.
Vegans going on and on repeating debunked onion pieces is very much like trying to debate with a flat earther. It doesn't matter how much evidence or science you use to show why the earth is not flat, they just believe it for whatever reason and no amount of common sense will change that.
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@LogicRoad @bigfatsurprise A reasonably high percentage of the population are cholesterol hyper absorbers (15-25%), so dietary cholesterol for them does impact plasma levels
radcliffecardiology.com/articles/under…
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@PattyW_1944 @SBakerMD The Lancet Commission 2024 released a list of the major risk factors for dementia - high LDL and hearing loss (equal first at 7%)
thelancet.com/journals/lance…

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@LLicit_Tweet @SBakerMD This looks like a recipe for Alzheimers, give me Carnivore over all that muck ..
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@PattyW_1944 @SBakerMD "Saturated fat intake, which typically increases on a ketogenic diet, is strongly associated with AD risk. In the Chicago Health and Aging Project, high saturated fat intake was linked to a 2- to 3-fold increased risk of incident Alzheimer's"
frontiersin.org/journals/nutri…
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@PattyW_1944 @SBakerMD "Conclusions: This study indicated that total dietary fat intake, especially 👉 saturated fat, contributed to the risk of Alzheimer's disease, and the effects were independent of other nutrients"
doi.org/10.1192/bjp.20…
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@bigfatsurprise Is Nina Teicholz right about saturated fat or is she a lobbyist for the meat industry?
youtube.com/watch?v=OkqWdY…

YouTube
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@bigfatsurprise No surprises here from Miss Meatfluencer
126,000 men & women followed for up to 32 years
Assessments every 4 years
Dietary fat: From foe to friend? SCIENCE16 NOV 2018 : 764–770

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