Crip Toe Hawaii

1/ Hawaii just introduced a new law targeting crypto kiosks (like Bitcoin ATMs). Here’s what it says and why it matters:🧵

@Shadalac2 @RippleXrpie I stand corrected 😂

@criptoehawaii @RippleXrpie They didn't learn youtube.com/shorts/fLO4aDQ…

🚨NO FKN WAY!! 🤯 The government admitted that they have faked Epstein’s suicide.

@helios_brah His content is literally next level. Takes me several days to dissect and absord the info.

fyi Kruse has been making content for 20ish years The bulk of it are his 900+ (!) long-form blogs Thats not even counting his stuff on Twitter and LinkedIn I made an estimate: A regular person would need like 2 months to get through this casually IF it were just normal text There is nothing even remotely normal about this text. Its 900+ articles of absolutely hardcore biophysics, 2-5 pages each (some 14 pages lol) with shitloads of slides and videos and just all kinds of shit Depending on how hard and/or deep you want to go here and what your background is Youre looking at A YEAR minimum, basically Thats an optimistic case for a Biochem PhD or Dr who is very open to things If youre a dickhead and think you know everything better (=everyone) you will need longer Good luck out there bros

@krakensupport @Abe_Blinkoln I want my 1099da!

@Abe_Blinkoln We apologize for the delays in the delivery of Form 1099-DA. We are issuing forms as quickly as our processes allow and have experienced technical challenges while ensuring accurate and secure delivery that protects your data privacy and reflects the information in your account.

@krakensupport where is my tax form????? Send it immediately to users. What a joke

@accounting @krakenfx Never got mine! I was forced to do my taxes without the 1099da

@krakenfx promised Form 1099-DA by 3/13. I’m hearing many haven’t received them. Are you still waiting on your 10999-DA?

@BorisJohnson Bitcoin is not a Ponzi scheme. A Ponzi requires a central operator promising returns and paying early investors with funds from later ones. Bitcoin has no issuer, no promoter, and no guaranteed return—just an open, decentralized monetary network driven by code and market demand.

@Shadalac2 @RippleXrpie The last time that worked was with Obama’s birth certificate. They learned their lesson so they dont use layers anymore

@RippleXrpie Anyone try the photoshop layer removal tool yet? Might get the unredacted version.

@edZ67011399 @ritarighteye @anomalie_blue Ive only had that problem with Mag citrate and oxide. Never with Mag Glycinate.

@criptoehawaii @ritarighteye @anomalie_blue Any magnesium of any form of any amount makes me violently shit myself

Melatonin which inhibits thyroid & progesterone is the 4th most popular “natural product” for adults. Imagine if people knew about glycine, salt, milk + honey…

@diglloyd @MetabolicUncle Interesting hypothesis. Why do you think it’s magnesium deficiency?

@MetabolicUncle A possible root cause (used to ride 10K miles a year) is chronic magnesium deficiency from ultra endurance training. No one studies this. They're all stupid as hell for not looking at the most relevant factor.

EXERCISE, PLAQUE, AND THE INCONVENIENT TRUTH NOBODY WANTS TO HEAR... THIS IS BRUTAL The most heavily trained athletes have almost six times more arterial plaque than those who train the least. That's not a typo. Six times. This contradicts everything the fitness industry sells you. But the data doesn't care about your feelings or your Strava stats. German researchers in 2008 examined 108 marathon runners over 50 who'd completed at least five marathons in three years. They matched them against controls with identical cardiovascular risk profiles. The only difference was the running. The marathon runners had more calcified plaque. Not less. More. Everyone assumed this was wrong. It had to be. Marathon runners are supposed to have pristine arteries. That's the whole point of cardiovascular exercise, right? The cardiovascular part. But then 2017 happened. Two papers in Circulation confirmed the pattern. UK Masters endurance athletes showed elevated plaque despite lower traditional risk factors. Another study found 77% of high-volume exercisers had arterial plaque versus 56% in low-volume exercisers. The only consolation was that the high-volume group's plaque was calcified, supposedly more stable, less likely to rupture and kill you. A small comfort when you've spent thousands of hours training specifically to avoid this outcome. Then 2023 arrived with worse news. The Masters at Heart Consortium studied 191 lifelong endurance athletes, 191 late-onset athletes, and 176 active controls. They screened out cardiovascular risk factors. Clean slate. Lifelong endurance athletes had significantly more plaque. But here's where it gets interesting. Unlike the 2017 findings, there was no protective calcification pattern. The plaque in lifelong athletes wasn't less dangerous than in sedentary people. It was just more abundant. So what drives this? Volume or intensity? Previous studies couldn't answer this because they relied on self-reported data. People recalling their training patterns, researchers converting memories into MET minutes per week. The numbers looked precise. They were essentially fiction. This new study used wearable monitors. Actual measurements. They calculated training load by multiplying duration by heart rate intensity. The result: highest intensity exercisers had almost six times the plaque risk. When researchers analyzed the same participants using traditional self-reported methods, the association vanished. The measurement method was everything. High intensity alone without volume didn't correlate with plaque. But high volume combined with high intensity showed strong connections. Not volume. Not intensity. The combination. Before you throw your running shoes in the trash, choose the sprint and intervall trainign methods that I regularily recommend. A study with 21,000 participants followed for 17 years found high-volume exercisers had more plaque markers but were not more likely to die from heart disease or any other cause. The plaque existed. It didn't kill them. So exercise increases plaque volume in high-volume athletes, but the plaque doesn't increase death rates. The downsides to cardiovascular health get offset by other benefits. This is where conventional wisdom fails you. Exercise is still the most powerful longevity tool available. Nothing in these studies changes that. Exercise still lowers all-cause mortality. Focus on hard outcomes like heart attacks and deaths, not surrogate markers like plaque scores. But fitness is not immunity. Being fit doesn't mean you get to ignore other cardiovascular risk factors. High-volume athletes can have significant plaque buildup. The solution isn't to stop exercising. The solution is to train smarter to get even more out of excercise and actually prolong your life expectancy! The fitness industry won't tell you this. They'd rather sell you another recovery supplement. But the data shows that extreme exercise volume combined with high intensity creates a specific cardiovascular stress pattern that manifests as increased plaque formation. This doesn't mean exercise is bad. It means exercise is a tool with specific effects, some beneficial, some neutral, some potentially problematic in certain contexts. Understanding these nuances lets you optimize outcomes instead of blindly following dogma.

@upgradejames So fat-free would be even better?

For as long as humans have been skimming cream to make butter, we have consumed low fat milk Whole milk is designed to grow a calf into a full sized cow. It’s an extremely anabolic food by nature, this can be a double edged sword. If you're trying to optimise glucose oxidation and hit your calcium requirements without becoming obese, skimmed is far superior Benefits i’ve noticed: - allows me to effortlessly hit 2g+ of calcium without encountering the Randle cycle - look and feel leaner, denser and less inflamed (gain much of the anabolic properties without getting fat) - digests with ease and is way more hydrating Skimming the fat also removes many of the issues associated with conventional dairy (like fat soluble endocrine disruptors and homogenisation), so it can make cheap and accessible option for those who don’t have access to higher quality dairy (watch out for additives).

@BigolWave Even fat free milk?

A lot of people don't know that there's a lot of stuff in both milk and orange juice that lowers estrogen. For example milk has progesterone and thyroid hormone in it which both oppose estrogen.. and orange juice has naringin which also antagonizes estrogen

@hawaiian_x_ Pdai all the way

I'm going to get my first purchase on Pulsechain for this year. I think the community is down so much, these prices are literally stealing candy from a baby. What would you rather buy...... (no, I do not want to buy bitcoin or XRP. Don't even bother asking. I buy blood.

@esegbona_luis Cardi is a low quality woman. She wont attract any high quality men.

🚨 Breaking News 😳😳😳 Cardi Puts Offset and Stefon in the Same Box – "I'm Too Pretty to Be Played" 💔 Cardi B just used her stage to send a loud and clear message to both of her baby daddies – and the crowd ate it up. What She Said: During her show, Cardi grouped Offset and Stefon Diggs together, shading them both. Her message? She's too pretty to be played with and way above their league. "Guys out there are praying to have a beautiful and successful woman like me. Offset and Stefon played themselves – thinking it was me they played." The Ironic Twist: Meanwhile... she's now the baby mama to both of them.

@User97PB @questmoosa I do this also but not every day. You have to be careful of Cadmium content.

@questmoosa Apparently raw cacao powder is high in copper so i just put some of that in my coffee

If you supplement with 30 mg’s of zinc per day, then you should also take 3-6 mg’s of copper to balance out the zinc-induced copper depletion.

@wonder_blu @AJA_Cortes Did taking that large of a dose make you sleepy?

@AJA_Cortes i took extreme high doses when healing from stage III colon cancer. i am a big believer in adding this to cancer protocols. i elected to forgo chemo and took a different path and am - knock on wood - doing great 2.5 years out….. (high doses = 1000mg/day)

Dr. Russel Reiter has been studying the properties and benefits of melatonin for 60 years He's 90 years old Still teaches and publishes research at UT San Antonio Takes 100mg at a time

@DrJackKruse This is next level mind blowing stuff and I actually understand it. I will re-read it a few times to fully grasp all the details. Thanks Jack

Look for 15 years I have tried to educate you. THIS IS THE LAST TIME. So pay attention. Your Rockefeller education beliefs are all FALSE. When dueterium leakage occurs into the matrix it stimulates cataplerosis in the TCA and the M1 Phenotype results. This process leads directly to the CDR too because tunneling speeds are destroyed. Here is where you, Peter D and your Rockefeller education screwed you. You know shit about tunneling speeds. Quantum mechanics tells us according to the formula for tunneling, even a tiny increase in distance (measured in angstroms) leads to an exponential drop in energy efficiency and a massive spike in ROS = massive non coherent UPEs that are the hallmark of disease generation. this is why I showed you Picard picture on the change in IMJ geometry. When the IMJs change you know tunneling speeds have cratered. Deuterium's KIE is a massive problem for tunneling speeds. But you and your food guru friends have no idea why. Quantum tunneling is extremely sensitive to mass. Deuteriumhas double the mass of H+. In the quantum world, as scale shrinks the effects become logrithmic because of the inverse square law. This means the effect of deuterium is off the chart. The doubling the mass of the particle attempting to "tunnel" across a gap doesn't just slow it down, it makes the probability of a successful tunnel drop exponentially. This is not subject to your beliefs or your experts beliefs because these UNIVERSAL laws in physics true on Earth or another galaxy. The Result: Electrons and protons "stutter." This delay increases the "dwell time" of electrons on the respiratory chain, leading to their premature escape and the creation of Superoxide. This is a ROS. ROS makes UPEs. You have no clue what a UPE even is because you're a food guru. It is a biophoton. The ATP synthase, (FoF1), is a physical motor that spins at speeds up to 9,000 RPM. It is designed specifically for the "light" weight of a single proton. The "Wobble" Effect: When a deuterium atom enters the top of the motor, the extra mass creates a mechanical imbalance, much like a lead weight on a high-speed fan blade. The Breakdown: This "wobble" creates friction and heat, physically damaging the mitochondrial inner membrane and causing leakage of the proton gradient. This leakage is a primary signal that triggers CDR1, as the cell perceives a loss of "pressure" (voltage) and shifts into defense mode. Deuterium in the matrix then causes the 25(OH)D Crash. Remember the VDR sits on the IMM as a brake for unfettered ROS/RNS production. Mitochondria rely on metabolic water production at CCO which is deuterium depleted. It is a highly structured, liquid-crystalline state of H2O. This type of water excludes solutes and anything over the size of a H+. This includes isotopes like deuterium which are double the size/mass of H+. When any stressor hits the system, say like nnEMF light stress, nnEMF shrinks the EZ water layer through CCO dehydration . When this layer collapses, deuterium "leaks" into the matrix and the enzymes and causes the destruction. As deuterium replaces protium in the water shells surrounding the CYP450 enzymes, the vibrational frequency of the enzyme changes. The enzyme loses its "quantum tune." To compensate for the loss of mitochondrial efficiency caused by deuterium "pollution," the body consumes Vitamin D at a massive rate to try and dampen the resulting ROS and stabilize the membrane. Once deuterium pollutes the matrix: OXPHOS fails: The motors are broken. Cataplerosis is unleashed: The cell stops trying to make ATP and starts dumping TCA intermediates (like citrate) to create lipids for new membranes (to replace the damaged ones). UPEs spike: The "friction" from deuterium in the nanomotors releases incoherent photons (UPEs), signaling to neighboring cells that the environment is toxic. The unleashing cataplerosis should make you realize why the M1 (Pro-inflammatory) Phenotype is always the result: Because the TCA cycle breaks at two SPECIFIC places (at Isocitrate Dehydrogenase and Succinate Dehydrogenase). This causes a massive efflux (cataplerosis) of Citrate and Succinate into the cytosol. Succinate then acts as a potent inflammatory signal, stabilizing HIF-1α and further shutting down oxidative phosphorylation (OXPHOS). In this state, the mitochondria stop making "energy and hormones" and start making "defense signals and ROS." the ROS makes non coherent UPEs and this causes all the diseases of man. You do not know this because you do not read shit about biophotons. Van Wijk and Popp have written careers on it while you wasted your time with your Rockefeller biochemistr retard PhD who knows none of this. This explains why our Vitamin D receptor sits on the IMM because it is a key regulator of this entire process. Melanin sits on the skin in cholesterol rafts capturing UVB light to stop this process. That is why the SUN heals it all and most of your bullshit beliefs are all linked back to Rockefeller solutions in centralized biochemistry and BigHarma. The VDR on our IMM trys to act as a "circuit breaker" for the CDR tied to deuterium effects in the matrix. The "Sinister" Element of deuterium is an expansion of Mitochondrial Heteroplasmy = disease. See Doug Wallace. LEARN. ANY increase in heteroplasmy = mitochondrial DNA damage. nnEMF increases the distance between respiratory proteins on the inner mitochondrial membrane. This slows down Electron Tunneling Speed (e). According to the formula for tunneling, even a tiny increase in distance (measured in angstroms) leads to an exponential drop in energy efficiency and a massive spike in ROS = massive non coherent UPEs that are the hallmark of disease generation. In metabolic terms, cataplerosis is the process of "draining" or siphoning intermediates out of the Tricarboxylic Acid (TCA) cycle. Here is what that "unleashing" looks like in the body: Siphoning for Survival (Biosynthesis): Instead of intermediates completing the full cycle to generate ATP, they are exported to create essential building blocks. This happens in cancers. Citrate is pulled for fatty acid synthesis. Alpha-ketoglutarate is drained to make amino acids or neurotransmitters. Oxaloacetate is diverted toward gluconeogenesis to maintain blood sugar. This is why nnEMF raises blood sugar in stress response. Preventing "Clogging": Cataplerosis acts as a pressure-relief valve. If intermediates (anions) accumulate too heavily in the mitochondria, it can disrupt cellular function. Unleashing this process clears out the "sink" so the cycle can continue to process incoming fuel. The Cost of "Unleashing": Because the TCA cycle is a closed loop, removing these "rungs of the ladder" will eventually break the cycle unless they are replaced by a counter-process called anaplerosis (refilling the cycle). Environmental Link: In your specific context, light stress from a move and heavy nnEMF is unleashing cataplerosis might be the body’s way of prioritizing oxidative repair or calcium buffering over standard energy production. It’s a "emergency mode" where the body spends its metabolic currency (like Vitamin D and TCA intermediates) to fix immediate damage. It really tells you how bad the place you went to was. In short: it is your metabolism switching from "maintenance mode" to "construction/repair mode" because your body is sensing a cell danger response. What I am saying to you , is that your body "used what it needed in this moment," but it left a collateral wake in your system. What are the system collateral effects? I am likely describing a massive decoupling event at the Cytochrome P450 (CYP450) level. why? When Fe in in its +3 state it cannot bind oxygen and P450 cannot make hormones = pregnenlone steal syndrome. Hormone synthesis from cholesterol relies on the CYP450 (Cytochrome P450) family of enzymes, which are heme-containing proteins. The Power of Heme: Heme is the "business end" of these enzymes, acting as a specialized scaffold for electron transfer. To function, it requires precise quantum coherence, which is the ability of electrons to "tunnel" and move in a wave-like, coordinated state. UPE Disruption: Non-native EMFs (nnEMF) act as electromagnetic "noise" that disrupts this coherence. This triggers the production of non-coherent Ultra-weak Photon Emissions (UPEs). ROS and Heme Damage: The resulting flood of Reactive Oxygen Species (ROS) from the mitochondria (the "danger mode" or CDR1) is highly toxic to "free" or loosely bound heme. When the heme center of a CYP enzyme is damaged or its electron flow is decoupled by ROS, the enzyme can no longer convert cholesterol into pregnenolone (the "mother hormone"), effectively halting the entire steroidogenic pathway at the source. If the environmental signals (like high milligauss levels from nnEMF) are constantly triggering the CDR, the mitochondria stay stuck in a pro-inflammatory state (M1 phenotype) designed for defense, not repair. the M1 state is caused by non-coherent UPE signaling. This is why high nnEMF environments are deadly when you blow away your P450 system. True photorepair healing requires moving from CDR1 (defense) to CDR2 (growth) and finally CDR3 (differentiation and restoration). A "toxic" environment, one with high nnEMF, poor light cycles, or chemical stress, acts as a persistent "block" that keeps cells from completing this cycle. Mitochondrial Mosaics: Over time, staying in the same environment creates a "mosaic" of dysfunctional cells that have failed to complete the healing cycle, leading to chronic illness or reigniting the flame of disease. Lesson Over.

@whatimemetosay @TaraBull 😂

New low cut dress ad featuring couple goes viral

CONTEXT: Other states (like NY, Iowa, Illinois, Connecticut) are also discussing how to regulate or restrict prediction markets — but Hawaii’s committee is the first to advance a full ban.

NEXT STEPS: HB 2198 — now amended to guard against repeal until at least 2029 — moves next to the House Judiciary Committee as the state’s legislative session continues.

BREAKING: A Hawaii House committee unanimously advanced a bill (HB 2198) to ban prediction markets — making the state the first in the nation to take this step. #hawaii #crypto #laws #gambling #bitcoin