
Dr Murphy WeightLoss
198K posts

Dr Murphy WeightLoss
@TakeWeightOffMD
Board-certified: Internist, Obesity Med. Geneticist, RegenMed, Orthobiologist and DPC practice. Pro Paleo Doc! Author and Longevity Doc. using rapamycin etc.






WHO Health Emergencies Communication Manager Nyka Alexander explains how hantavirus could be managed in the UK as six people have been released from a Merseyside hospital and must now self-isolate



@john_malone It’s not malpractice when all the professional society guidelines say to prescribe after a 2 (at best 9) question survey that basically anyone would test “positive” for on any moderately tough day “But judge, that’s what the gUiDeLiNeS say” Case dismissed


1 in 3 college aged kids being on psych meds is absolutely bonkers We’ve completely lost the plot

An antiviral pill has, for the first time, been shown to prevent COVID-19 in people exposed to the SARS-CoV-2 virus go.nature.com/4nAMi6g






Endothelial dysfunction and metabolic biomarkers in post-COVID-19 syndrome 🚨A nightmare study for many! FOR EVERYBODY: 9 months after your COVID-19, your blood vessels are STILL damaged, with probably biomarkers to prove it! 1. Study design: German prospective cohort (n=262 adults) compared blood biomarkers ~37 weeks post-SARS-CoV-2 infection (PCS patients + recovered) versus uninfected controls. 2. Endothelial dysfunction: - Soluble thrombomodulin (sTM) and lactate dehydrogenase (LDH), two established markers of endothelial (blood-vessel) damage, were significantly elevated in the combined infected group (UOC PCS + LIFE Cov+) versus never-infected controls. - So, previously infected individuals had significantly higher soluble thrombomodulin (TM) and lactate dehydrogenase (LDH), independent of PCS diagnosis or fatigue severity. - Critically, there were no significant differences between the PCS group and the LIFE Cov+ group for these markers. 3. Metabolic disruption: - Clear alterations in arginine biosynthesis and taurine/hypotaurine pathways, indicating impaired nitric-oxide (NO) metabolism with reduced L-arginine and taurine levels. →In plain terms: the vascular damage signal was just as strong in the symptom-free recovered people as in those still suffering from fatigue and other PCS complaints. The same held for the metabolic disruptions (impaired arginine biosynthesis and taurine/hypotaurine pathways linked to nitric-oxide production). 4. Fatigue-specific signal: - High-fatigue PCS patients (top MFI-20 quartile) showed elevated linoleic acid (LA) plus monounsaturated fatty acids oleic (OA) and palmitoleic (PA). →The fatty-acid signature specific to high-fatigue PCS adds a potential severity marker. 5. Notes: - Changes persisted already up to 9 months after infection. - PCS remains symptom-based, but these markers offer objective, measurable signals of underlying vascular and metabolic damage! -The discussion and results treat the observed endothelial and metabolic changes as consequences of a single infection and do not speculate on additive damage from subsequent exposures. - Limitations well noted. ‼️So, COVID-19 leaves detectable, objective blood-vessel and metabolic damage in the bloodstream nearly a year later, even if you feel completely fine and never had Long COVID symptoms. Specific fatty-acid spikes flag severe post-COVID fatigue. The “it’s all in your head” narrative for many post-covid people is directly contradicted by these biomarkers! These findings, solidly backed by prior 2021–2025 evidence, prove that COVID-19 inflicts measurable, persistent endothelial and nitric-oxide metabolic damage up to nearly a year later, even in fully symptom-free survivors, thereby supporting the implementation of routine biomarker screening, but even more importantly: #AvoidSars2 #AvoidReinfections. YOU ARE NOW A CV RISK PATIENT! nature.com/articles/s4159…









