Philippe Rola

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Philippe Rola

Philippe Rola

@ThinkingCC

#zentensivist, EMCrit Teammate, Proud daddy and husband. BJJ🟪. ICU Santa Cabrini Hospital.

Montreal, Canada Tham gia Ağustos 2013
1.1K Đang theo dõi11.7K Người theo dõi
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Pierre Poilievre
Pierre Poilievre@PierrePoilievre·
Le ministre libéral François-Philippe Champagne affirme s'être récusé du projet de TGV en septembre 2025. Un projet de 90 milliards $ dont sa conjointe est la vice-présidente. Pourtant, il en a fait la promotion lors d'un témoignage au Sénat six mois plus tard. Dans le budget qu'il a lui-même présenté, on ne retrouve aucune trace de sa récusation et il a aussi voté en faveur du TGV à la Chambre des communes. La version du ministre Champagne ne tient pas la route. 90 milliards $ sont en jeu. Vous devez dire la vérité, monsieur le ministre.
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Jonathan Kay
Jonathan Kay@jonkay·
It increasingly looks like Canada will be (literally) the last place on the planet where parents who think their kid was “born in the wrong body” can come to get the child sterilized, drugged, & surgically disfigured to conform to their cultish beliefs nationalpost.com/opinion/canada…
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Philippe Rola
Philippe Rola@ThinkingCC·
Nope. We didn’t think it was enough. Wt-based approach never made sense. Only DP makes sense. We believe strategy should involve minimizing it and getting back to FRC! @EMNerd_ @DoctorDaxon @khaycock2 @DrMiguelIbarra1
Dr. Chacón-Lozsán F .'.@franciscojlk

🫁 We thought “lung-protective ventilation” was enough. It isn’t. ⚠️ The problem Low tidal volume (4-8 ml/kg PBW) changed outcomes. But here’s the issue: 👉 ARDS is not one lung 👉 It’s a patchwork of different lungs Collapsed units Overdistended areas Normal regions 👉 Same ventilator setting = completely different stress per region 🧠 The key concept: “Baby lung” You’re not ventilating 5 liters. You’re ventilating: 👉 maybe 1-2 liters of functional lung So even “safe” tidal volumes can become: 🔥 Overdistension in disguise 💥 What actually causes VILI? Not just pressure. Not just volume. 👉 It’s uneven distribution of stress and strain Four classic mechanisms: Barotrauma Volutrauma Atelectrauma Biotrauma But now we understand: 👉 Mechanical power = the real integrator ⚡ The new paradigm Ventilation is not about settings...It’s about physiology. 🔬 What should we actually target? 💡 1. Driving pressure (ΔP) 👉 Best bedside surrogate of lung stress 👉 Aim <15 (ideally <10) 💡 2. Mechanical power 👉 Energy delivered to the lung 👉 High MP = higher mortality 💡 3. Recruitability 👉 Does PEEP open lung or overdistend it? 💡 4. Patient effort 👉 Too much → P-SILI 👉 Too little → diaphragm atrophy 💡 5. Regional ventilation (not global) 👉 EIT, LUS, esophageal pressure = game changers 🫀 The part we ignore too often Ventilation is NOT just lungs. 👉 It affects: Right ventricle Cardiac output Kidney perfusion Brain function 👉 VILI = multi-organ disease 🤖 The future is already here Closed-loop ventilation AI-driven adjustments Digital twins of the lung 👉 From reactive → predictive ventilation 🚨 Take-home message > There is no “safe ventilator setting” There is only a setting that is safe for THAT patient, at THAT moment 🎯 Final thought We need to move from: ❌ Protocol-driven ventilation To ✅ Physiology-guided precision ventilation 📚 Reference Merola, R., Battaglini, D., Schultz, M. J., & Rocco, P. R. M. (2026). Physiology-guided personalized mechanical ventilation to prevent ventilator-induced lung injury. Frontiers in Medicine, 13, 1764151. doi.org/10.3389/fmed.2…

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Philippe Rola
Philippe Rola@ThinkingCC·
@max_berrill @IM_Crit_ @Srivatsa34 The Praxis project is coming soon. The AI coaches will be free, we are building quite an educational community. I had intended to launch last week, but the coaches still need a little more fine-tuning so I am anticipating launching on May 1.
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IMCrit
IMCrit@IM_Crit_·
Right ventricular (RV) involvement complicates >30% of acute inferior STEMIs, though hemodynamically significant RV infarction occurs in ~10–15% of cases. The classic teaching is that when a patient w inferior STEMI becomes hypotensive & has clear🫁on physical exam, the treatment
IMCrit@IM_Crit_

ICU - Board Review Qs: 60 yo pt admitted to the ICU because of inferior STEMI. Emergency cath: 100% proximal RCA occlusion treated successfully with stenting One hour post-PCI: dyspnea/anxiety - BP: 94/70, HR: 60/min (sinus). Phys exam: JVD (+), clear lungs, cool extremities

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Philippe Rola
Philippe Rola@ThinkingCC·
@dravukati Yup also an option to help dbp, but as you know this won’t help the RV directly so may combine w inotropy. Of course as was pointed out cath has to r/o occlusion.
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Uzman Hekim Görüşü
Sir even a basic mechancial support like IABP would be most helpful in this scenario. Rv shock is treated first with revascularisation and second heamodynamic support. Normally lv coronary supply is dependant on diastolic drive but rv coronary can fill with systolic drive also so augmenting output and bp without too much stress on rv is crucial.
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Musstdu Daswissen 
@wkcmd @ThinkingCC Sometimes when you consider Dobutamin+Norepi the answer might be Epi- even if you don’t want tachycardia and you don’t want to increase O2 consumption of the myocardium. It might still be the answer- low low low continuous dose
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Philippe Rola
Philippe Rola@ThinkingCC·
@ImagenCardiaca Yes, of course I understand not many are using it yet, but I always find it odd when authors refer to something that is already well established in the literature as a measure of coupling as being something novel…
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SEIC - Sociedad Española de Imagen Cardíaca
Querido @thinkingCC : is grounded in physiological principles described in the 1970s–90s (e.g., the work of Karel H. Sunagawa), but its real emergence is recent: over the last decade—and especially in the past 2 years—it has been incorporated into clinical cardiology through echocardiography. In this sense, we consider it “new”: new in our daily practice.
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Matt Siuba
Matt Siuba@msiuba·
Come join us in FL this summer to learn about critical care hepatology Virtual option available too with discounted rates for trainees Agenda and signup link in 🧵
Matt Siuba tweet media
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Philippe Rola
Philippe Rola@ThinkingCC·
@pjcotera @YubSedhai Totally agree that has to be the first thing that needs to be ruled out, but I was going with the aim of the question that seemed to be about haemodynamic management.
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Pablo Cotera
Pablo Cotera@pjcotera·
@ThinkingCC @YubSedhai If suspicion is acute stent thrombosis wouldnt dobutamine in an hypoxic RV just increase RV O2 consumption and increase infarction size? No real right answer IMO. Would rush back to cath.
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Philippe Rola
Philippe Rola@ThinkingCC·
@MynephCC Absolutely! I’ve been burned by this so many times, very frustrating. A quick super pubic peek with any probe should be automatic in any patient with a Foley catheter, especially one who is intubated and sedated, and cannot complain!
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Ahmed T Abdellah
Ahmed T Abdellah@MynephCC·
Confirmation of Foley catheter placement is essential in patients with gross hematuria, especially prior CBI. While initial urine return may suggest correct placement, POCUS in this case demonstrated a persistently distended bladder with a large clot no Foley's in the bladder.
Ahmed T Abdellah tweet media
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Philippe Rola
Philippe Rola@ThinkingCC·
Just putting a feeler out there...Anyone up for #VExUSinMilan (Italy) next november? (did I mention participants will get to meet @khaycock2 in the flesh?)
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Yub Raj Sedhai, MD
Yub Raj Sedhai, MD@YubSedhai·
@ThinkingCC I fully understand the concept, I was just highlighting the fact that are failure in setting of proximal RCA MI, and acute and chronic RV systolic dysfunction on a background of chronic pulmonary hypertension are two different physiological constructs!!
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Katrina (Trina) Augustin
Katrina (Trina) Augustin@TrinaAugustinMD·
RAPID ECPR: Revolutionizing Accelerated Percutaneous Initiation and Deployment of ECPR: single center process and outcomes - Journal of Cardiothoracic and Vascular Anesthesia jcvaonline.com/article/S1053-…. Excited to finally share this data, really proud of the incredible Mayo team!
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