mjash

A truck with the confederate flag printed on its hood was part of Saturday's Winter Carnival parade in downtown Saranac Lake.

Want to see your fat burning go over 1g/min or even higher? Reduce carb intake substantially. Muscle glycogen content at the start of exercise sets the rate of fat oxidation during subsequent exercise. Muscle glycogen determines its own rate of oxidation by directing the rate of fat oxidation. The key effect of this regulation is to replace muscle glycogen use with fat oxidation when muscle glycogen content is low and the reverse when muscle glycogen content is high. The more carb eaten before or during exercise, the less fat and the more glycogen that will be burned during exercise. It's the exact opposite of what we've taught over the years. And the reason for our error was we failed to realise that glucose use during exercise is split into 2 compartments that are separately regulated: The small glucose pool in the liver, intestine and blood stream and which supports the blood glucose concentration; and the large glucose pool in the skeletal muscles. Metabolism in the large glucose pool is always directed to burn muscle glycogen as the priority fuel if it is present. Muscle burns even more glycogen if carbs are ingested, since they reduce muscle fat oxidation causing the muscles to substitute glycogen for fat oxidation. We also have subconsciously accepted the notion that the main goal of human metabolic design is to produce fast running humans running exclusively on carbs during high intensity exercise. That is, the human is a machine designed principally to run fast by using carbs to produce the required energy. But the key priority in human metabolic design it to protect the blood glucose concentration, not to fuel up for faster running. When these 2 priorities conflict, regulation of blood glucose control always wins. And blood glucose regulation is directed by the small glucose pool; not the large pool. This has the unforseen consequences described above.