Russell Long

@Mylovanov Can't be correct... those wheels alone weigh more than 26 lbs.

At a secret factory in southern Germany, Helsing SE mass-produces AI attack drones for Ukraine. The HX-2 weighs 26 pounds, can cost as little as €17,500, needs barely a week of training and has been deployed by the thousands, NYT. 1/

@PhilipJohnston When brilliance comes knocking, only a fool says "no". You and your team are awesome... thanks for your outstanding work... it's gonna help cap terrestrial emissions, too... a big f deal for the planet. 🌏🌍🌎🔥

Hahah we now get the same regarding our preseed at the end of 2023 (slide below) where investors that passed now say “Oh, if I’d know you were going to build thiiss…” I used to finish every pitch with this slide and say all compute is going to space once the launch cost hits a certain point. Some seem to have forgotten! For context, we were raising $2M at $10M cap SAFE and had at least 100 investors say no. Thanks @FinnMurphy12 for leading and @russelllong for being the first ‘yes’!

@Angry_Staffer Well, Russia has orchestrated political hit jobs before leaving virtually no trace. Lindsey would have been number one on their list right now. I hope FBI does a full analysis.

Nobody can tell whether the FBI is assisting with the investigation into Lindsey Graham’s death because they actually suspect foul play or if Kash just wants attention. It’s pretty pathetic.

@_hrmsel @CaolanReports Troll.

@CaolanReports Why don't you go to the frontline if you love Ukrainians so much?

This week Kyiv was hit by missiles & Irish politicians are STILL defending the refinery supplying Russia’s war machine. Today, another ship sailed from Ireland to Russia. For years, the government tried to cover this up. Now the truth is coming out Watch my full investigation:

@ChrisMartzWX You're rude and obnoxious. Try to be more respectful of others. Muting you.

Quiet, piggy. 🐖 Nighttime temperatures have nothing to do with heatwaves. Heatwave frequency, intensity, and their duration have always historically been evaluated using daily Tmax at / above the 90th percentile for 3–6 consecutive day periods (TX90p). Heatwave frequency, intensity, and duration have decreased in the U.S. since the start of the 20th century. Facts don’t care about your feelings, you retarded, inbred vegetable. Try again.

@nntaleb @bryan_johnson You're being reactionary. My Dad took 90 supplements a day and lived past 90. Maybe without them he could have lived to 95 but give me a break... Bryan is allowing us all to be part of his experiment which is generous, especially when trolls attack, like yourself.

MATH QUIZ DUJOUR @bryan_johnson (a.k.a. Draculino) takes 100 pills a day plus other crazy interventions. A very sad story. 1) How many possible interactions between pairs of drugs? 2) How many for 3 drugs? 3) And generalizing, how many higher order interactions (n drugs)?

@ralfduschef @r0ck3t23 There’s a reason why we read respected journalists inthe New York Times instead of watching a bunch of self-appointed “ influencers” with credulous followers.

@r0ck3t23 In telecoms they talk about pipe and service. Inasfar as media are pipe they are commodity and doomed. But Marc is over-optimistic: Media also do impedance-matching, service. An expert-builder may well not be a good teacher or explainer. You have to have the receiver in mind too.

Marc Andreessen just named the lie that held for a hundred years and is collapsing in real time. We gave authority to the people who explained things. Not the people who built them. And nobody questioned why. For a century, builders created and journalists translated. The public accepted it because complexity demanded a middleman. But the middleman was never the expert. The middleman was the channel. And we mistook the channel for the source. Andreessen: “You set it loose and it will write you literally a 30-page answer. This is basically like a textbook on any topic.” Any topic. Infinite depth. Zero cost. No gatekeeper. He has a name for what comes next. Practitioner media. Andrej Karpathy doesn’t sit across from a journalist. He turns on a camera and teaches the world how the architecture works. No filter. No editorial framing. No one deciding what you’re ready to hear. The press calls it dangerous. They are not protecting the public. They are protecting the bottleneck that gave them power. The critic always needed the creator. The creator never needed the critic. They just had no other way to reach the world. Now they do.

@igorsushko If true, why isn’t this generating more mainstream media attention? It’s an incredible number.

💥 Ukraine struck 10 more Russian fuel tankers today in the Azov Sea on the way to refuel occupied Crimea. Russia has lost 48 such ships in total in the Azov Sea over 5 days, including tugs and support vessels. Around the number of 1588 Spanish Armada ships lost to the storm.

@PhelanArt @JakeLFischer Warriors top offer was 3 years, $75m with team option, but Kuminga instead chose a two-year deal to retain more control over his future, turning it down; the Warriors also had a standing two-year, $45M offer and a three-year, $54M fully guaranteed alternative.

@JakeLFischer He has an awful, greedy, delusional agent. They turned down 5 years $150 million from Warriors. He is worth $12 million per year, TOPS! Should be fired...

To be clear: Said on yesterday’s show Jonathan Kuminga is looking for “more in” the $25 million AAV ballpark compared to the Lakers’ previously-reported 2/20 offer. Not “more than.” And Kuminga is also open to various deal structures depending on fit and role with Lakers, Cavs, other suitors. Full detail on his sign-and-trade market: tinyurl.com/d8a6n7u9

@gnnaali15 @bryan_johnson Someone's gonna do it, one day. Why not Bryan, why not now?

@bryan_johnson Your passion is nice but it’s laughable that you think you’ll cure an autoimmune disease when thousands of scientists with PhDs haven’t yet cured it 😂

My plan to cure autoimmune gastritis To our knowledge, no one has ever done this to try and cure an autoimmune disease. Context: In May, I got diagnosed with autoimmune gastritis (AIG). We found it by taking a tissue biopsy of my stomach. My immune cells are confused, causing my stomach to eat itself. AIG stops your body from absorbing nutrients like iron and B12, and can eventually lead to cancer. It likely started decades ago when I was diagnosed with hypothyroidism when 21 years old. The thyroid and stomach are closely linked in your immune system. I feel fortunate that I've been taking such good care of my body for the past five years as my condition would otherwise be much more severe. Millions of people are affected by this disease and are undiagnosed. Standard of care tells you that you can’t do anything about it. That’s old fashioned. Here is how we are going to try and cure it: Step 0: find and diagnose the disease ✅ AIG is rarely caught early because symptoms are subtle. Early warnings are low iron and B12, but when hemoglobin and hematocrit look normal, doctors routinely miss it because there are no obvious signs of anemia. A standard colonoscopy won't find it either, because it only checks the lower digestive tract, not the stomach. It was only through a highly targeted stomach biopsy that we found it. Even biopsies can miss it if they don't sample the exact right spots. Most people with AIG go undiagnosed. Step 1: Map my immune system ✅ Last Thursday, I had a blood draw to isolate and decode 1 million of my immune cells. Think of your immune cells as trillions of soldiers. Each carries a unique key designed to unlock and destroy a specific threat, like a virus or bacteria. A standard blood test allows you to see how many soldiers you have, but not their keys. Sequencing one million individual immune cells allows us to read the exact pattern of the teeth on every single key. This is important for my autoimmune gastritis (AIG) because a specific platoon of rogue soldiers has developed keys that unlock an attack on my stomach lining. Right now, we don’t know who they are. This test will inform us of which soldiers have gone rogue and are attacking me from within. Once we know the soldier and key, we know what therapy path to pursue to shut them down. Step 2: Catch the rogue soldiers I will be getting a second biopsy from my stomach because we need to collect live tissue. We are currently planning out the logistics of getting the sample from my stomach to the lab. We need these live cells because the initial blood tests showed the antibodies, which prove that an attack is happening, but doesn’t show us the actual rogue soldier doing the damage which is a T-cell. The live sample will allow us to match the immune system mapping we did to the live T-cells. Step 3: Build an early warning system To keep an eye on the disease as we work towards a therapy, we’re building an early warning system. I'll have my blood drawn every two weeks and we’ll pair that information with wearable data to look for flare ups. This is important because the attack happens without producing symptoms that I can easily feel. Step 4: Create a “Bryan in a dish” testing model, a miniature of my immune system At the same time, we are taking a massive sample of my immune cells and deep freezing them (cryopreservation) for two reasons: a) we’ll create a living lab: using these cells to replicate my immune environment in a lab dish. This allows us to test experimental drugs and therapies on my actual live cells before putting them into my body. b) it creates a back up plan for me by preserving the raw cellular material needed for targeted rejuvenation therapies in the future. Step 5: Build precision guided therapies to end the attack Once we know who the rogue soldiers are, we will engineer a therapy designed uniquely for them. The trick is only turning off the rogue soldiers while leaving all the other healthy ones functioning as they are. For safety checks, we’ll do two test runs: 1) we’ll run the therapy through a computer model that has my biology to evaluate how my molecules interact. 2) We will take my actual cells that we froze in Step 4 and watch them interact for real. If both are successful, we’ll pursue one of four therapies: a) fix the mistake my cells are making, restoring my immune system's natural off switches b) teach the rogue cells to tolerate my stomach instead of attacking it c) design smart molecules that physically plug into the rogue cells and turn them off d) build soldiers who will track down and eliminate the rogue soldiers causing the damage

@norsegaud @bryan_johnson Yikes. How did they eventually find the virus?

+ it could be pointing to a more serious problem + try your preferred method to correct: meat, supplements, etc. I actually disagree with this. I had a family friend who was hospitalized for low iron, they gave him supplements, and turns out a virus in his liver was using iron to grow and ended up getting out of control and killing him.

I had low iron for 11 years. Yes, even when I ate meat, my ferritin was low and averaged 38 ng/mL. I’ve finally boosted it back to healthy levels. You can see my protocol below. On the surface, my low ferritin was easy to dismiss by most standards of care. Most doctors miss this or don't investigate it deeply enough, including mine during those years. I get it. The human body is complicated and the science is constantly evolving. My hemoglobin and hematocrit were normal. Ferritin measures stored iron, while hemoglobin measures circulating iron, and because the body drains its reserves first to keep hemoglobin normal, you can be fully iron deficient with a perfectly normal hemoglobin and hematocrit. This is why my low ferritin kept getting dismissed: the numbers that define anemia looked fine, so no one asked why my iron reserves wouldn't refill. During these 11 years, before doing health stuff and when on my longevity protocol, I sadly did not fully understand how important iron was to my body. Now, everywhere I look, I see iron playing a central role. Still, during those 11 years, I tried everything to fix it, including eating meat and every type of oral iron supplementation, using every timing trick and every formulation. None of the iron would stick and we couldn’t figure out why. I overhauled my medical team earlier this year. With greater capacity, we revisited everything. Then we discovered my autoimmune gastritis (AIG). With AIG, my stomach doesn't make enough acid to absorb iron. The only route left was to bypass the gut and deliver it intravenously. Most people treat anemia as the threshold to watch. But long before hemoglobin drops, low iron starves the enzymes your cells depend on: the ones your mitochondria use to make energy, synthesize DNA, build dopamine and other neurotransmitters, and power immune defense. That is why you can feel fatigue, brain fog, and worse endurance even with normal hemoglobin and an otherwise "normal" iron panel. We did a deep dive on possible iron infusion therapies. I ended up getting a 1000mg monoferric infusion. Why we chose Monoferric: > Monoferric is more tolerable, allowing a higher dose of up to 1000 mg > a complete replenishment dose in one infusion > other IV irons require multiple infusions (3-5) > head-to-head randomized trials show it causes hypophosphatemia in only about 8% of patients compared to 74% with Injectafer > its most common side effects are mild nausea and rash occurring in roughly 1% of patients > other options, including Iron dextran (INFeD), carries a black-box FDA warning for potentially life-threatening allergic reactions and requires a mandatory test dose before each new treatment course > it's very expensive. Your doctor can write a letter to your insurance company to justify medical necessity. Ferritin levels post infusion: + 205 ng/mL 2 weeks post infusion + 195 ng/mL 4 weeks post infusion Our target is 80 ng/mL. We will continue to monitor. Levels are expected to settle around 6-8 weeks post-infusion. The lessons I’ve learned: + don’t mess with low iron + don’t accept it as ok + it could be pointing to a more serious problem + try your preferred method to correct: meat, supplements, etc. + and if it doesn’t correct, investigate why, and consider an infusion

@AndroidParanorm @bryan_johnson So? If he succeeds in disclosing, and solving a previously hidden problem, Big Pharma will pass along all costs to the government -- the higher the better for them -- and the rest of us will finally get access.

@bryan_johnson Unsurprisingly, this infusion is something that only the top 0.1% of income earners would be able to afford this treatment on a regular basis.

@bryan_johnson Excellent write-up and analysis. Thanks so much for sharing it. I'm there with you on autoimmunity (Graves/Hash/LADA Type-1), and will run my own gastric/parietal labs to see if that's why my own ferritin runs low without red meat. Sadly, MDs are still terrible with this stuff.

@bryan_johnson Bryan, I also have Graves/Hashimotos and low ferritin. I started iron supplements that drove iron to the top of range and ferritin barely moved (22-->45). Had to reduce supplemental iron and ferritin dropped again. The solution was to add red meat. Have you tried this approach?

Bad news #1: I have an autoimmune disease. My stomach is eating itself. Bad news #2: 2–5% of people have this, too. Likely more, because it hides. Good news: I'm going to try and solve it. Will share all. As a kid, I ate sugar cereal, drank sugary soda, and gobbled down fast food. I had a few healthy years in my early 20s but then became a young father of three and began building a business. Juggling that stress and grind, I let my health slip and gained 40 lbs. Within a few years I’d fallen into a deep, chronic depression. Somewhere in that timeline, my body began developing an autoimmune process affecting my thyroid and then my stomach lining. It’s called Autoimmune Gastritis (AIG). My hypothyroidism got diagnosed when I was 21 years old with a routine blood draw. That enabled me to begin proactive management, supplementing levothyroxine and Armour Thyroid. They are the hormones my body should be producing on its own but wasn’t. By taking these pills daily, my body was able to operate as though my thyroid was functioning properly. What I didn’t know was that something else was going on inside my body: my stomach had begun attacking itself. But there was no routine test to find out and I didn’t have any symptoms. I just discovered it in May. I'm unsure how long I've had it. AIG causes irreversible damage: nutritional deficiency, anemia, and over a long horizon, elevated cancer risk. When AIG is discovered today, standard medical care concedes defeat, stating that nothing can be done except managing the condition, no matter how awful or lethal the effects. Looking back over the past few years, I can now see the early signals we were picking up in measurement but hadn’t connected the dots. For 11 years, I’ve had low ferritin, without anemia. We continually tried to raise my iron levels with food and supplementation but nothing would work. We chased the obvious solutions first. A plant-based diet means all my iron is the hard-to-absorb, non-heme kind. Hard training, sauna, and hyperbaric oxygen all raise the body's demand for iron. But none of them explained the core failure: despite me taking iron orally, trialing every formulation, and using every timing trick, none of the iron would stick. What I didn’t fully appreciate until recently is how many stones my previous providers had left unturned. The low ferritin kept getting explained away but not fixed. I overhauled my medical team earlier this year. It was the rebuild to lay the groundwork for Immortals Care, our $1M a year protocol. With greater capacity, we revisited everything. On the surface, my low ferritin was easy to dismiss by most standards of care. My hemoglobin and hematocrit were normal. Ferritin measures stored iron, while hemoglobin measures circulating iron, and because the body drains its reserves first to keep hemoglobin normal, you can be fully iron deficient with a perfectly normal hemoglobin and hematocrit. This is why my low ferritin kept getting dismissed: the numbers that define anemia looked fine, so no one asked why my iron reserves wouldn't refill. My team pressed on that question. They first turned to a colonoscopy. I was 48 years old and overdue. It was good health hygiene to have while also serving a specific purpose of searching for a hidden source of blood loss such as a polyp or even cancer in my bowels. Either one of those would be an explanation of why the iron kept disappearing. At the same time, they began connecting the dots. Iron absorption depends on stomach acid, so one theory was that my stomach acid was disrupted. They also knew that thyroid and stomach autoimmunity often travel together, so often that the pairing has a name: thyrogastric syndrome. Put against my 27+ year history of autoimmune thyroid disease, the pieces pointed to a single hypothesis: my own immune system was attacking my stomach. To our surprise, my colonoscopy came back clean. A perfectly healthy colon, better than 95% of colonoscopies of men, according to the gastroenterologist. That ruled out the first concern and worst possible outcome: slow continuous bleeding from colon cancer, or pre-cancerous polyp. My team had exercised great foresight though, anticipating this possible outcome. In addition to a colonoscopy, they’d ordered an upper endoscopy to be performed at the same time. The combined procedure is a bi-directional endoscopy. Probes would look at my entire intestinal tract, up from below and down the throat. Additionally, we had several blood biomarkers measured ahead of the procedure to try and pick up on any signals that would give the gastroenterologist guidance for what to look for while doing visual inspections. Fifteen minutes before the procedure, my blood results returned, finding elevated levels of anti-parietal-cells-antibodies (APCA). They came back at roughly five times the upper limit of normal (103, against a ceiling of 20 Units/mL). It was a positive result confirming the suspicion of AIG being the culprit behind my low ferritin, the other type of gastritis, driven by a bacterial infection, was already ruled out, as we knew I am negative to H. pylori. Even before this finding, my team had ordered five biopsies to be taken from three regions of my stomach. The biopsies were the critical piece. Had they not been ordered, the bi-directional endoscopy would have been completed and AIG remained undiagnosed as there were no visual signatures of the condition in my intestines. Two days later, the results of biopsies came in, showing clear signs of early autoimmune gastritis: early atrophy confined to the acid-producing lining, with the rest of the stomach still spared. My team had anticipated this, methodically tracing every line of evidence. We now had a formal diagnosis. I have autoimmune gastritis AIG. My stomach is eating itself. So this was never one problem. It was three, linked to one another: the iron deficiency, the autoimmune gastritis driving it, and the autoimmune thyroid disease alongside it. Iron and thyroid feed each other both ways, low iron impairs the conversion of thyroid hormone into its active form, and an under active thyroid impairs how the body uses iron. Each made the other harder to fix. Autoimmune gastritis affects an estimated 2–5% of people, and likely more, because it hides and is challenging to diagnose. It's usually silent for years, surfacing only once the stomach has atrophied enough to do real damage: iron deficiency first, then B12 deficiency, then anemia from both, and over a long horizon, raised stomach-cancer risk. In one study of people with precancerous gastric lesions, roughly 18% carried the autoimmune antibodies, and only about 1% had ever been diagnosed. And the earliest clue, low ferritin, is the one standard medicine waves through. Low iron stores get normalized and rarely investigated at all when anemia hasn't shown up yet. That blind spot is what hid mine for a decade. The good news: the iron deficiency is now corrected. I received a 1,000 mg Monoferric iron infusion. This was chosen for two reasons after considering multiple formulations. First, it can safely deliver a full dose of iron in a single infusion (1,000 mg), while older options like Venofer require several separate appointments to reach the same total. Second, certain other IV iron formulations can cause a drop in blood phosphate levels, an important mineral for bones and energy. Monoferric is much less likely to do this, which matters given how closely we track long-term metabolic and bone health parameters. As mentioned earlier, current medical standards treat AIG as something to be managed, not resolved. It's worth noting that many of you give me a hard time, inviting me to "live life" and engage in self-destructive behaviors like a "normal person". I'm cool with the playful ribbing. Also, had I not taken care of my health during the past five years, my situation could potentially be very serious. You too may have a lurking health issue that is undiagnosed and could increase in severity from unhealthy life choices, without your knowing. The absence of symptoms is not the presence of health. A gentle nudge that minding your health, no matter your situation in life, is good decision making. My team and I are going to try and solve my AIG. This is how we’re approaching it: First, routine monitoring keeps the disease in view: ferritin and iron, B12, the pepsinogen I/II ratio, gastrin, and chromogranin A. Gastrin is the dial to watch. If it climbs, the disease is advancing, and the risk of gastric neuroendocrine tumors climbs with it. Second, we’re doing advanced characterization of the disease. We’ll do a repeat biopsy to read the immune infiltrate, deep cytokine profiling, and T-cell subset analysis, to see which pathways are actually firing. That testing drives the intervention plan, including the experimental approaches we intend to develop. + If gastrin and chromogranin rise: damp the gastrin drive (netazepide) and tighten endoscopic surveillance. If the profile is Th1 / interferon-driven: target JAK/STAT. + If it's Th17 / IL-17-driven: target IL-17 and STAT3. + If regulatory T cells are failing: rebuild them (low-dose IL-2, induced Tregs). + If it's antibody- and B-cell-driven and antigen-specific: engineered cell therapy (CAAR-T). Which organizes into four tiers, from available today to frontier: Tier 1, now: protect and support; zinc-L-carnosine, and acid replacement (betaine HCl with pepsin) under physician supervision. This is specific to my case and not something to self-prescribe, especially given the cancer-surveillance considerations above. Tier 2, target the signaling , JAK/STAT, GSK-3, IL-17, and damp the gastrin drive (netazepide). Tier 3, reset the cells, induced regulatory T cells (iTregs). Tier 4, frontier: engineered T-cell therapy (CAR-T / CAAR-T), custom AI-designed antibodies, or synthetic proteins, that can specifically seek out inactivate or destroy the rogue immune cells attacking my stomach lining. To be clear: there's no approved cure for autoimmune gastritis today. Medicine treats it as something to manage, not solve. Tiers 2 through 4 are investigational preclinical evidence at best, and in several cases therapies that still have to be built. If you're working on autoimmune gastritis, antigen-specific tolerance, regulatory T cells, or CAAR-T for organ-specific autoimmunity, please reach out. Modern medicine has normalized too many conditions that erode our health, function, and comfort, shrinking the goal to monitoring and management while a cure is rarely even attempted. Most of these verdicts were handed down decades ago, in an era that predates nearly all of our current tech and science, and they have gone largely unchallenged. We want to change that. In the age of AI, multiomics, and custom-built DNA, proteins, and cells, no condition should be presumed incurable simply because no one has yet tried to cure it with today's stack. I’ll end on a personal note. We fill our days mostly on things that are trivial next to what we ultimately care about. We know, deep down, however, that in the noise of it all, health is easily forgotten until it’s the only thing that matters. We spend a fraction of our lives truly sober to the preciousness of life. We feel it when someone we love dies, when a child is born, when we come close to death ourselves, or when a diagnosis marks our limit. In those moments, we are sobered, and the rarity of it all becomes self evident. Imagine the existence we’d build together if that clarity didn’t fade. I wish all of you the very best. Care for yourself, care for others, care for the planet and care for our animal friends. Care for life as it’s the most precious gift there is.

@sportsguy_rich @ChrisGloninger Call your Republican legislators! They are still doing whatever Trump wants.

@ChrisGloninger Call Al Gore!

The oceans just hit the hottest temperature ever recorded. 90%+ of the heat we trap by burning fossil fuels goes into the ocean - the clearest fingerprint of what's happening. And it broke during the cool phase. El Niño is only just arriving. #ClimateChange #OceanHeat #ElNino

@hausfath Yikes. I guess ski season is over before it starts. More worrisome though are droughts/famines in Africa/India....

July ECMWF runs are in and forecast an even more extreme El Niño event than the prior June runs. ENSO3.4 region temperatures reach 3.9C in December, and forecasts are around 0.3C warmer than they were in June.

@LegendsMadden @ShamsCharania @anthonyVslater You're not wrong and I like this kid, but $10m/yr is a lot when you've already got $20m into Porzingis, $7m into Horford, and have options with Bassey and Graham Ike too.

Yall warrior fans are sooo fucking retarted if you think this isnt fair, post shot 40% from 3 his rookie year but he was a cone on defense like fucking abysmal, this year his shot went down but he was top 7 in defensive rating when he was on the floor and statistically one of the best defensive bigs in the league and even watching him he met the eye test he was a substantially better defender at the 5 than draymond. The only reason post shot dipped this season was because he was putting alot of effort defensively and his legs wasn’t strong enough to handle both ends of the floor. All he has to do is work on his legs this summer and post will be a starting caliber 2 way big thats a legitimate floor spacer especially in the west vs wemby. My only gripe with him is his soft rebounding but he was literally getting better at rebounding as the season progressed but kerr wasnt playing him much towards the end not only due to injury but because his shot wasnt falling. Not only that but dude has the hunger to play, mans sprained his ankle and rather than sitting out and letting it heal he taped that bitch up and wanted to hoop, dumbass fanbase wants to get rid of every player just because they dont have star potential 🤦🏽 NOT EVERY PLAYER NEEDS TO BE A STAR TO BE GOOD

Restricted free agent Quinten Post is signing a three-year, $30 million contract offer sheet with the Memphis Grizzlies, sources tell ESPN. The Golden State Warriors have until 11:59pm ET on Tuesday to match.

On this hot fourth of July it is worth reminding folks that its not the sun that's causing global warming. Solar output (as measured by satellites) has been flat over the past 50 years when global temperatures have shot up. Its our emissions of CO2 and other greenhouse gases.

@atrupar Clickbait. There is NO nervous breakdown.

here is the entirety of Palantir CEO Alex Karp's televised nervous breakdown this morning on CNBC

@PhilipJohnston @benchmark @Starcloud_ @EverettRandle @chetanp @MollySOShea @sourceryy 💯

“The @Benchmark investment in @Starcloud_ was about the people.” Thanks @EverettRandle and @ChetanP Puttagunta 🙏🥹 @MollySOShea, @sourceryy